diff --git a/BACKEND/INSTALL/QUICKSTART/README.md b/BACKEND/INSTALL/QUICKSTART/README.md index 0d4d307..bd04077 100644 --- a/BACKEND/INSTALL/QUICKSTART/README.md +++ b/BACKEND/INSTALL/QUICKSTART/README.md @@ -52,7 +52,7 @@ While still in $TG_DIR/BACKEND/INSTALL, please execute the following command: biorels_php gen_single_script.php > biorels_execute.sh sh biorels_execute.sh -This will execute the necessary scripts in the correct order. +This will execute the necessary scripts in the correct order. It will take about an hour and a half. ## Querying the database @@ -76,3 +76,4 @@ Please go to $TG_DIR/BACKEND/SCRIPT/BIORJ/ You can execute the following scripts: * biorels_php api_import.php --JSON Example/HeLa.biorj * biorels_php api_export.php -JSON_OUTPUT=CVCL_B6YM.biorj --JSON_PRETTY_PRINT CELL CVCL_B6YM && more CVCL_B6YM.biorj +* biorels_php api_import.php --JSON Example/rs712.json diff --git a/BACKEND/INSTALL/biorels_public.sql b/BACKEND/INSTALL/biorels_public.sql index 50ca054..8f20ce1 100644 --- a/BACKEND/INSTALL/biorels_public.sql +++ b/BACKEND/INSTALL/biorels_public.sql @@ -3272,14 +3272,14 @@ COMMENT ON COLUMN DB_SCHEMA_NAME.org_group_map.org_group_id IS 'Foreign key to o COMMENT ON COLUMN DB_SCHEMA_NAME.org_group_map.web_user_id IS 'Foreign key to web_user representing a scientist'; CREATE SEQUENCE DB_SCHEMA_NAME.org_group_map_sq - START WITH 3822421 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE CACHE 1; CREATE SEQUENCE DB_SCHEMA_NAME.org_group_sq - START WITH 75516 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE @@ -5121,7 +5121,7 @@ COMMENT ON COLUMN DB_SCHEMA_NAME.source.source_type IS 'Type of database/resourc COMMENT ON COLUMN DB_SCHEMA_NAME.source.source_metadata IS 'Additional information'; CREATE SEQUENCE DB_SCHEMA_NAME.source_seq - START WITH 1194 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE @@ -5420,8 +5420,7 @@ CREATE TABLE DB_SCHEMA_NAME.variant_frequency ( variant_change_id bigint NOT NULL, variant_freq_study_id smallint NOT NULL, ref_count integer NOT NULL, - alt_count integer NOT NULL, - source_id smallint + alt_count integer NOT NULL ); COMMENT ON TABLE DB_SCHEMA_NAME.variant_frequency IS 'Frequency in different studies of a given allele'; @@ -5436,10 +5435,9 @@ COMMENT ON COLUMN DB_SCHEMA_NAME.variant_frequency.ref_count IS 'Reference count COMMENT ON COLUMN DB_SCHEMA_NAME.variant_frequency.alt_count IS 'Total count'; -COMMENT ON COLUMN DB_SCHEMA_NAME.variant_frequency.source_id IS 'Foreign key to source.'; CREATE SEQUENCE DB_SCHEMA_NAME.variant_frequency_sq - START WITH 18778296255 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE @@ -5659,7 +5657,7 @@ CREATE SEQUENCE DB_SCHEMA_NAME.web_job_document_sq CACHE 1; CREATE SEQUENCE DB_SCHEMA_NAME.web_job_sq - START WITH 1640 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE @@ -5734,7 +5732,7 @@ COMMENT ON COLUMN DB_SCHEMA_NAME.web_user.manager_id IS 'Foreign key to web_user COMMENT ON COLUMN DB_SCHEMA_NAME.web_user.worker_type IS 'Type of worker - employee or contingent'; CREATE SEQUENCE DB_SCHEMA_NAME.web_user_seq - START WITH 2000 + START WITH 1 INCREMENT BY 1 NO MINVALUE NO MAXVALUE @@ -7390,6 +7388,9 @@ ALTER TABLE ONLY DB_SCHEMA_NAME.variant_freq_study ALTER TABLE ONLY DB_SCHEMA_NAME.variant_frequency ADD CONSTRAINT variant_frequency_pkey PRIMARY KEY (variant_frequency_id); +ALTER TABLE ONLY DB_SCHEMA_NAME.variant_frequency + ADD CONSTRAINT variant_frequency_uk UNIQUE (variant_change_id, variant_freq_study_id); + ALTER TABLE ONLY DB_SCHEMA_NAME.variant_info ADD CONSTRAINT variant_info_pkey PRIMARY KEY (variant_info_id); diff --git a/BACKEND/INSTALL/runInstall.php b/BACKEND/INSTALL/runInstall.php index e50500f..e5c6add 100644 --- a/BACKEND/INSTALL/runInstall.php +++ b/BACKEND/INSTALL/runInstall.php @@ -4,16 +4,22 @@ $TG_DIR=getenv('TG_DIR'); if ($TG_DIR===false)die('TG_DIR not set in setenv.sh');/// TG_DIR is not set as environment variable if (!is_dir($TG_DIR))die('Unable to find directory '.$TG_DIR);//// TG_DIR is not an existing directory -if (!is_file($TG_DIR.'/BACKEND/CONTAINER/env-file.txt'))die('No environment file found'); -if (!is_file($TG_DIR.'/BACKEND/CONTAINER/biorels_container.sif'))echo ('Warning - No container file found'); +if (!is_file($TG_DIR.'/BACKEND/CONTAINER/env-file.txt'))die('No environment file found'."\n"); +if (!is_file($TG_DIR.'/BACKEND/CONTAINER/biorels_container.sif'))echo ('Warning - No container file found'."\n"); + +$AGREE_ALL=false; +if (isset($argv[1]) && $argv[1]=='--agree') $AGREE_ALL=true; + echo "Root directory for biorels: ".$TG_DIR."\n"; -$resSTDIN=fopen("php://stdin","r"); +if (!$AGREE_ALL) +{ + $resSTDIN=fopen("php://stdin","r"); echo("Do you confirm? Y/N. Then press return: "); $strChar = stream_get_contents($resSTDIN, 1); if ($strChar=='N') die('You did not agreed'."\n"); if ($strChar!='Y') die('We didn\'t understood the answer'."\n"); - +} /// Getting environment variables defined in setenv.sh. Here it's DB_SCHEMA, the name of the public schema $DB_SCHEMA=getenv('DB_SCHEMA'); if ($DB_SCHEMA===false) die('No schema provided'); /// Getting environment variables defined in setenv.sh. Here it's PRIVATE_SCHEMA, the name of the private schema @@ -22,12 +28,14 @@ echo "\n\n\nPublic schema:".$DB_SCHEMA."\n"; +if (!$AGREE_ALL) +{ $resSTDIN=fopen("php://stdin","r"); echo("This will delete this schema if it exists. Do you confirm? Y/N. Then press return: "); $strChar = stream_get_contents($resSTDIN, 1); if ($strChar=='N') die('You did not agreed'."\n"); if ($strChar!='Y') die('We didn\'t understood the answer'."\n"); - +} echo "\n\nCreating tables for ".$DB_SCHEMA."\n"; /// Here we are going to convert the sql template file into the installation file, which includes the correct schema name. @@ -47,11 +55,14 @@ if ($PRIVATE_SCHEMA!==false) { echo "\n\n\n\nPrivate schema:".$PRIVATE_SCHEMA."\n"; + if (!$AGREE_ALL) + { $resSTDIN=fopen("php://stdin","r"); echo("This will delete this schema if it exists. Do you confirm? Y/N. Then press return: "); $strChar = stream_get_contents($resSTDIN, 1); if ($strChar=='N') die('You did not agreed'."\n"); if ($strChar!='Y') die('We didn\'t understood the answer'."\n"); + } $change=array('DB_SCHEMA_NAME'=>$DB_SCHEMA,'DB_PRIVATE_SCHEMA'=>$PRIVATE_SCHEMA); //// Here we convert the template sql file by changing the template schema name to the private schema name convertFile($TG_DIR.'/BACKEND/INSTALL/biorels_private.sql',$TG_DIR.'/BACKEND/INSTALL/private_schema_ready.sql',$change); diff --git a/BACKEND/SCRIPT/API/queries.php b/BACKEND/SCRIPT/API/queries.php index d180c70..68cdc02 100644 --- a/BACKEND/SCRIPT/API/queries.php +++ b/BACKEND/SCRIPT/API/queries.php @@ -2774,14 +2774,12 @@ function get_translation_isoform_pos($ISOFORM_ID,$POSITION) # // Title: Search gene ontology record by name and/or accession and/or namespace # // Function: search_gene_ontology # // Description: Search for a gene ontology record by using its name and/or accession and/or namespace -# // Parameter: AC | Gene ontology accession | string | GO:0010389 | optional +# // Parameter: AC | Gene ontology accession | string | GO:0010389 # // Parameter: NAME | Gene ontology name | string | cell cycle | optional # // Parameter: NAMESPACE | Gene ontology namespace | string | biological_process | optional # // Return: Gene ontology record # // Ecosystem: Proteomics:gene ontology # // Example: php biorels_api.php search_gene_ontology -AC 'GO:0010389' -# // Example: php biorels_api.php search_gene_ontology -NAME 'cell cycle' -# // Example: php biorels_api.php search_gene_ontology -NAMESPACE 'biological_process' # // $[/API] function search_gene_ontology($AC='',$NAME='',$NAMESPACE='') { diff --git a/BACKEND/SCRIPT/BIORJ/BIORJ_RULES b/BACKEND/SCRIPT/BIORJ/BIORJ_RULES index 5b379ea..fa11bf1 100644 --- a/BACKEND/SCRIPT/BIORJ/BIORJ_RULES +++ b/BACKEND/SCRIPT/BIORJ/BIORJ_RULES @@ -155,14 +155,15 @@ CHILD drug_atc_mapclinical_variant_pmid_mapclinical_variant_disease_mapclinical_variant_gn_mapvariant_changevariant_changevariant_change>variant_frequencyG","last_submitted_date":"2021-07-13"}],"pmid_entry":[{"pmid_entry_id":20915747,"pmid":21688344,"publication_date":"2012-05-01 00:00:00","title":"Effects of KRAS mutation and polymorphism on the risk and prognosis of oral squamous cell carcinoma.","doi":"10.1002\/hed.21792","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":4450,"month_1910":-1228,"full_text_date":null},{"pmid_entry_id":21030132,"pmid":21811665,"publication_date":"2011-01-01 00:00:00","title":"Clinical Significance of MicroRNA Expression Profiles and Polymorphisms in Lung Cancer Development and Management.","doi":"10.4061\/2011\/780652","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":25795,"month_1910":-1212,"full_text_date":null},{"pmid_entry_id":21361969,"pmid":22166496,"publication_date":"2012-03-01 00:00:00","title":"The role of microRNA-binding site polymorphisms in DNA repair genes as risk factors for bladder cancer and breast cancer and their impact on radiotherapy outcomes.","doi":"10.1093\/carcin\/bgr300","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":3888,"month_1910":-1226,"full_text_date":null},{"pmid_entry_id":22370049,"pmid":23293655,"publication_date":"2012-01-01 00:00:00","title":"Mechanistic insights into the role of microRNAs in cancer: influence of nutrient crosstalk.","doi":"10.3389\/fgene.2012.00305","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":26472,"month_1910":-1224,"full_text_date":null},{"pmid_entry_id":22774059,"pmid":23729275,"publication_date":"2013-10-01 00:00:00","title":"A let-7 binding site polymorphism rs712 in the KRAS 3' UTR is associated with an increased risk of gastric 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Neck","issn_print":"1043-3074","issn_online":"1097-0347","iso_abbr":"Head Neck","nlmid":"8902541"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":69914259,"pmid_entry_id":20915747,"pmid_author_id":8533283,"position":5},{"pmid_author_map_id":69914258,"pmid_entry_id":20915747,"pmid_author_id":19394246,"position":4},{"pmid_author_map_id":69914257,"pmid_entry_id":20915747,"pmid_author_id":7915151,"position":3},{"pmid_author_map_id":69914256,"pmid_entry_id":20915747,"pmid_author_id":7964073,"position":2},{"pmid_author_map_id":69914255,"pmid_entry_id":20915747,"pmid_author_id":19054519,"position":1}],"pmid_abstract":[{"pmid_abstract_id":18062362,"pmid_entry_id":20915747,"abstract_type":"BACKGROUND","abstract_text":"Mutations or single nucleotide polymorphism (SNP) of relevant genes may affect the risk and prognosis of malignancies. The purpose of this study was to analyze whether the KRAS polymorphisms and mutations can be useful prognostic or risk markers in oral squamous cell carcinoma (OSCC)."},{"pmid_abstract_id":18062363,"pmid_entry_id":20915747,"abstract_type":"METHODS","abstract_text":"DNA was extracted from tumor tissues of 47 patients with OSCC and blood cells of 84 normal controls and subjected to sequencing for the KRAS."},{"pmid_abstract_id":18062364,"pmid_entry_id":20915747,"abstract_type":"RESULTS","abstract_text":"No mutation in the KRAS was found in 47 OSCC samples. However, 2 polymorphisms (rs1137282 and rs712) were detected. Individuals with KRAS SNP rs712 genotypes of G\/T or T\/T have a reduced risk for OSCC than those with genotype G\/G (hazard ratio [HR], 0.26; 95% confidence interval [CI], 0.10-0.60; p = .004). The overall survival between different SNPs were not statistically significant (p = .147 for rs1137282 and p = .202 for rs712)."},{"pmid_abstract_id":18062365,"pmid_entry_id":20915747,"abstract_type":"CONCLUSION","abstract_text":"These data demonstrate a role for rs712 polymorphism of the KRAS in susceptibility of OSCC."}]},{"pmid_author":[{"pmid_author_id":7915151,"last_name":"Wong","first_name":"Yong-Kie","initials":"YK","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0f9ae10ca1291802a5cedbe7148aecf5"},{"pmid_author_id":7964073,"last_name":"Chien","first_name":"Yi-Chih","initials":"YC","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"5faac041bfd5834eaf45707017653e89"},{"pmid_author_id":8533283,"last_name":"Lin","first_name":"Jin-Ching","initials":"JC","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0d599a5f7b450c7c72fdce8666128211"},{"pmid_author_id":19054519,"last_name":"Wang","first_name":"Wen-Yi","initials":"WY","is_valid_author":"Y","pmid_instit_id":7262903,"orcid_id":null,"md5_hash":"b8f2ce03dbdc7568df0eaaecd601787a"},{"pmid_author_id":19394246,"last_name":"Lin","first_name":"Yan-Liang","initials":"YL","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"24708b673838011e22e3a2f6dc858e8f"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":7262903,"instit_name":"Section of Basic Medicine, Department of Nursing, Hung Kuang University, Taichung, Taiwan.","instit_hash":"f2dca89885f527db7eb4f61c78844ffa"}]}]},{"RECORD":{"pmid_entry_id":21030132,"pmid":21811665,"publication_date":"2011-01-01 00:00:00","title":"Clinical Significance of MicroRNA Expression Profiles and Polymorphisms in Lung Cancer Development and Management.","doi":"10.4061\/2011\/780652","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":25795,"month_1910":-1212,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":25795,"journal_name":"Pathology research international","journal_abbr":"Patholog Res Int","issn_print":"2090-8091","issn_online":"2042-003X","iso_abbr":"Patholog Res Int","nlmid":"101526292"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":70489091,"pmid_entry_id":21030132,"pmid_author_id":7791496,"position":3},{"pmid_author_map_id":70489090,"pmid_entry_id":21030132,"pmid_author_id":7790558,"position":2},{"pmid_author_map_id":70489089,"pmid_entry_id":21030132,"pmid_author_id":19566270,"position":1}],"pmid_abstract":[{"pmid_abstract_id":18248068,"pmid_entry_id":21030132,"abstract_type":"Text","abstract_text":"Lung cancers account for a huge percentage of death in industrialized countries, and hence there is an increasing call for the development of novel treatments. These malignancies are caused by a combination of environmental factors, principally cigarette smoking and genetic alterations. MicroRNAs (miRNAs) are a recently discovered class of regulatory noncoding small RNAs with a significance in numerous biological processes. Strong evidence links miRNA impaired expression profiles and pathways to the etiology of several diseases, including neoplasia. This paper focuses on the emerging role of miRNA function in lung cancer development with particular highlighting on the use of miRNA profiles and polymorphisms for the molecular and biological characterization of tumor pulmonary growth and progression. Furthermore, we underline the potential utility of lung cancer-associated miRNAs as clinical biomarkers with a diagnostic, prognostic, and therapeutic significance and give emphasis to the promising novel miRNA-based curative strategies."}]},{"pmid_author":[{"pmid_author_id":7790558,"last_name":"Pizzuti","first_name":"Antonio","initials":"A","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"9f950cc1e98d54a4364afd912d66f12c"},{"pmid_author_id":7791496,"last_name":"Frati","first_name":"Luigi","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"2cb887eebe7aff6b660fd376db61dcab"},{"pmid_author_id":19566270,"last_name":"Megiorni","first_name":"Francesca","initials":"F","is_valid_author":"Y","pmid_instit_id":7499992,"orcid_id":null,"md5_hash":"a189dcb3dc80d7a8b9f2b89f461cf440"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":7499992,"instit_name":"Department of Experimental Medicine, Sapienza University of Rome, Viale Regina Elena, 324-00161 Rome, Italy.","instit_hash":"57f9d54578868fc11d52d29872cb739c"}]}]},{"RECORD":{"pmid_entry_id":21361969,"pmid":22166496,"publication_date":"2012-03-01 00:00:00","title":"The role of microRNA-binding site polymorphisms in DNA repair genes as risk factors for bladder cancer and breast cancer and their impact on radiotherapy outcomes.","doi":"10.1093\/carcin\/bgr300","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":3888,"month_1910":-1226,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":3888,"journal_name":"Carcinogenesis","journal_abbr":"Carcinogenesis","issn_print":"0143-3334","issn_online":"1460-2180","iso_abbr":"Carcinogenesis","nlmid":"8008055"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":72130629,"pmid_entry_id":21361969,"pmid_author_id":9621503,"position":10},{"pmid_author_map_id":72130628,"pmid_entry_id":21361969,"pmid_author_id":8754965,"position":9},{"pmid_author_map_id":72130627,"pmid_entry_id":21361969,"pmid_author_id":8534086,"position":8},{"pmid_author_map_id":72130626,"pmid_entry_id":21361969,"pmid_author_id":8032071,"position":7},{"pmid_author_map_id":72130625,"pmid_entry_id":21361969,"pmid_author_id":10547568,"position":6},{"pmid_author_map_id":72130624,"pmid_entry_id":21361969,"pmid_author_id":16544633,"position":5},{"pmid_author_map_id":72130623,"pmid_entry_id":21361969,"pmid_author_id":14098105,"position":4},{"pmid_author_map_id":72130622,"pmid_entry_id":21361969,"pmid_author_id":8243999,"position":3},{"pmid_author_map_id":72130621,"pmid_entry_id":21361969,"pmid_author_id":11769122,"position":2},{"pmid_author_map_id":72130620,"pmid_entry_id":21361969,"pmid_author_id":20062383,"position":1}],"pmid_abstract":[{"pmid_abstract_id":18794335,"pmid_entry_id":21361969,"abstract_type":"Text","abstract_text":"MicroRNAs (miRNAs) are involved in post-transcriptional regulation of gene expression through binding to messenger RNAs (mRNA) thereby promoting mRNA degradation or altered translation. A single-nucleotide polymorphism (SNP) located within a miRNA-binding site could thus alter mRNA translation and influence cancer risk and treatment response. The common SNPs located within the 3'-untranslated regions of 20 DNA repair genes were analysed for putative miRNA-binding sites using bioinformatics algorithms, calculating the difference in Gibbs free binding energy (\u0394\u0394G) for each wild-type versus variant allele. Seven SNPs were selected to be genotyped in germ line DNAs both from a bladder cancer case-control series (752 cases and 704 controls) and 202 muscle-invasive bladder cancer radiotherapy cases. The PARP-1 SNP rs8679 was also genotyped in a breast cancer case-control series (257 cases and 512 controls). Without adjustment for multiple testing, multivariate analysis demonstrated an association with increased bladder cancer risk with PARP1 rs8679 (P(trend) = 0.05) while variant homozygotes of PARP1 rs8679 were also noted to have an increased breast cancer risk (P = 0.03). In the radiotherapy cases, carriers of the RAD51 rs7180135 minor allele had improved cancer-specific survival (hazard ratio 0.52, 95% confidence interval 0.31-0.87, P = 0.01). This is the first report of associations between DNA repair gene miRNA-binding site SNPs with bladder and breast cancer risk and radiotherapy outcomes. If validated, these findings may give further insight into the biology of bladder carcinogenesis, allow testing of the RAD51 SNP as a potential predictive biomarker and also reveal potential targets for new cancer treatments."}]},{"pmid_author":[{"pmid_author_id":8032071,"last_name":"Hall","first_name":"Janet","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"99f2d31abc87624a7d99d3f787ada495"},{"pmid_author_id":8243999,"last_name":"Taylor","first_name":"Claire F","initials":"CF","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"55303ce5b35dd8eb4913aaf103a239a3"},{"pmid_author_id":8534086,"last_name":"Landi","first_name":"Stefano","initials":"S","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"55f953df5048736142b1b8e663c017bb"},{"pmid_author_id":8754965,"last_name":"Bishop","first_name":"D Timothy","initials":"DT","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0e3901611c0186a3317bdec40ff16478"},{"pmid_author_id":9621503,"last_name":"Kiltie","first_name":"Anne E","initials":"AE","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"6b68e2e4a443e798804430a522ead8ce"},{"pmid_author_id":10547568,"last_name":"Cox","first_name":"David G","initials":"DG","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"e94e887219781707fb49037c35c89409"},{"pmid_author_id":11769122,"last_name":"Landi","first_name":"Debora","initials":"D","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"e43a01ca879c29385f3e1eb7c9b746ad"},{"pmid_author_id":14098105,"last_name":"Elliott","first_name":"Faye","initials":"F","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"f9101bd072bc1dff223cbe5a839428ac"},{"pmid_author_id":16544633,"last_name":"Vaslin","first_name":"Laurence","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"fdf1e06f410dbfe53c15d224338a3f29"},{"pmid_author_id":20062383,"last_name":"Teo","first_name":"Mark T W","initials":"MT","is_valid_author":"Y","pmid_instit_id":6273181,"orcid_id":null,"md5_hash":"e525bd7c09e47f270b8c599bb39fe52a"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":6273181,"instit_name":"Section of Epidemiology and Biostatistics, Leeds Institute of Molecular Medicine, University of Leeds, Leeds LS9 7TF, UK.","instit_hash":"f4a0d7a825acc043a3c11f2a4c906d58"}]}]},{"RECORD":{"pmid_entry_id":22370049,"pmid":23293655,"publication_date":"2012-01-01 00:00:00","title":"Mechanistic insights into the role of microRNAs in cancer: influence of nutrient crosstalk.","doi":"10.3389\/fgene.2012.00305","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":26472,"month_1910":-1224,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":26472,"journal_name":"Frontiers in genetics","journal_abbr":"Front Genet","issn_print":"","issn_online":"1664-8021","iso_abbr":"Front Genet","nlmid":"101560621"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":77279192,"pmid_entry_id":22370049,"pmid_author_id":8321218,"position":3},{"pmid_author_map_id":77279191,"pmid_entry_id":22370049,"pmid_author_id":8024999,"position":2},{"pmid_author_map_id":77279190,"pmid_entry_id":22370049,"pmid_author_id":21565735,"position":1}],"pmid_abstract":[{"pmid_abstract_id":20474379,"pmid_entry_id":22370049,"abstract_type":"Text","abstract_text":"A plethora of studies have described the disruption of key cellular regulatory mechanisms involving non-coding RNAs, specifically microRNAs (miRNA) from the let-7 family, the miR-17 family, miR-21, miR-143, and the miR-200 family, which contribute to aberrant signaling and tumor formation. Certain environmental factors, such as bioactive dietary agents, e.g., folate, curcumin, polyunsaturated fatty acids, are also thought to impact the progression and severity of cancer. In terms of the chemoprotective mechanisms of action, these bioactive dietary agents appear to act, in part, by modulating tissue levels of miR-16, miR-17 family, miR-26b, miR-106b, and miR-200 family miRNAs and their target genes. However, the mechanisms of nutrient action are not yet fully understood. Therefore, additional characterization of the putative underlying mechanisms is needed to further our understanding of the biology, early diagnosis, prevention, and the treatment of cancer. For the purpose of elucidating the epigenetic landscape of cancer, this review will summarize the key findings from recent studies detailing the effect of bioactive dietary agents on miRNA regulation in cancer."}]},{"pmid_author":[{"pmid_author_id":8024999,"last_name":"Davidson","first_name":"Laurie A","initials":"LA","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"4a23b4051a305ba4ff23f9728ea6cdc3"},{"pmid_author_id":8321218,"last_name":"Chapkin","first_name":"Robert S","initials":"RS","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c9f9703854adcb62532018305d2a8c93"},{"pmid_author_id":21565735,"last_name":"Shah","first_name":"Manasvi S","initials":"MS","is_valid_author":"Y","pmid_instit_id":8425087,"orcid_id":null,"md5_hash":"665ebedef137cf1cd0f729d1ae35de83"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":8425087,"instit_name":"Program in Integrative Nutrition and Complex Diseases, Texas A&M University, College Station TX, USA ; Intercollegiate Faculty of Genetics, Texas A&M University, College Station TX, USA.","instit_hash":"c5c4a2578a71110c9267cc0a42fa5ff8"}]}]},{"RECORD":{"pmid_entry_id":22774059,"pmid":23729275,"publication_date":"2013-10-01 00:00:00","title":"A let-7 binding site polymorphism rs712 in the KRAS 3' UTR is associated with an increased risk of gastric cancer.","doi":"10.1007\/s13277-013-0885-x","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":4046,"month_1910":-1245,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":4046,"journal_name":"Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine","journal_abbr":"Tumour Biol","issn_print":"1010-4283","issn_online":"1423-0380","iso_abbr":"Tumour Biol","nlmid":"8409922"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":79379023,"pmid_entry_id":22774059,"pmid_author_id":14856752,"position":7},{"pmid_author_map_id":79379022,"pmid_entry_id":22774059,"pmid_author_id":7845254,"position":6},{"pmid_author_map_id":79379021,"pmid_entry_id":22774059,"pmid_author_id":7741052,"position":5},{"pmid_author_map_id":79379020,"pmid_entry_id":22774059,"pmid_author_id":20137637,"position":4},{"pmid_author_map_id":79379019,"pmid_entry_id":22774059,"pmid_author_id":19921645,"position":3},{"pmid_author_map_id":79379018,"pmid_entry_id":22774059,"pmid_author_id":15717237,"position":2},{"pmid_author_map_id":79379017,"pmid_entry_id":22774059,"pmid_author_id":22190588,"position":1}],"pmid_abstract":[{"pmid_abstract_id":21170685,"pmid_entry_id":22774059,"abstract_type":"Text","abstract_text":"Recently, single nucleotide polymorphisms in let-7 miRNA binding site in 3' untranslated region (UTR) of KRAS mRNA have been found to be associated with the cancer risk. In this study, we genotyped the frequency of KRAS rs712 to test its effect on gastric cancer (GC) risk in a hospital-based case-control study in a Chinese population, with 181 histologically confirmed GC patients and 674 cancer-free controls, using polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP) assay. The TT genotype of rs712 was associated with an increased risk of GC when taking GG genotype as a reference (adjusted odds ratio (OR) = 3.05, 95 % confidence interval (CI), 1.53-6.08). Similarly, the T allele of rs712 was associated with a statistically significant increase in susceptibility compared with G allele (adjusted OR = 1.44, 95 % CI, 1.10-1.90). Our data demonstrated that the T allele of the let-7 binding site polymorphism rs712 in KRAS 3' UTR was associated with a significantly increased risk of GC, suggesting that the KRAS rs712 polymorphism may be a genetic marker for the development of GC."}]},{"pmid_author":[{"pmid_author_id":7741052,"last_name":"Zhang","first_name":"Zhen","initials":"Z","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"18ac4bd06dd04dd5cdb7532b1ec1d459"},{"pmid_author_id":7845254,"last_name":"Jia","first_name":"Jing","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c3fe88ec64f15b20fb4a77c6e1513715"},{"pmid_author_id":14856752,"last_name":"Gao","first_name":"Lin-Bo","initials":"LB","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8c034d9c5eb3949b6774cb771bf3c390"},{"pmid_author_id":15717237,"last_name":"Pan","first_name":"Xin-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"3cae845fe3f69767f0414c3dcc97b4de"},{"pmid_author_id":19921645,"last_name":"Han","first_name":"Bao-Wei","initials":"BW","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"a627dc1d18bb2fc09b264287d9ffdf2c"},{"pmid_author_id":20137637,"last_name":"Guo","first_name":"Xiao-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"bf51cfc4b6899c387c1b6bad4c651b08"},{"pmid_author_id":22190588,"last_name":"Li","first_name":"Zhao-Hui","initials":"ZH","is_valid_author":"Y","pmid_instit_id":8716776,"orcid_id":null,"md5_hash":"8103fa5a5a1c2763b408129e825760af"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":8716776,"instit_name":"Secondary Department of General Surgery, Luoyang Central Hospital Affiliated to Zhengzhou University, Luoyang, Henan, 471003, People's Republic of China.","instit_hash":"6e7b01d3bb143516d5b9991a124ad4ed"}]}]},{"RECORD":{"pmid_entry_id":23005534,"pmid":23975373,"publication_date":"2014-01-01 00:00:00","title":"A let-7 KRAS rs712 polymorphism increases colorectal cancer risk.","doi":"10.1007\/s13277-013-1114-3","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":4046,"month_1910":-1248,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":4046,"journal_name":"Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine","journal_abbr":"Tumour Biol","issn_print":"1010-4283","issn_online":"1423-0380","iso_abbr":"Tumour Biol","nlmid":"8409922"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":80560535,"pmid_entry_id":23005534,"pmid_author_id":14856752,"position":8},{"pmid_author_map_id":80560534,"pmid_entry_id":23005534,"pmid_author_id":13192794,"position":7},{"pmid_author_map_id":80560533,"pmid_entry_id":23005534,"pmid_author_id":19477763,"position":6},{"pmid_author_map_id":80560532,"pmid_entry_id":23005534,"pmid_author_id":7741052,"position":5},{"pmid_author_map_id":80560531,"pmid_entry_id":23005534,"pmid_author_id":20137637,"position":4},{"pmid_author_map_id":80560530,"pmid_entry_id":23005534,"pmid_author_id":10784523,"position":3},{"pmid_author_map_id":80560529,"pmid_entry_id":23005534,"pmid_author_id":21341765,"position":2},{"pmid_author_map_id":80560528,"pmid_entry_id":23005534,"pmid_author_id":22570289,"position":1}],"pmid_abstract":[{"pmid_abstract_id":21567181,"pmid_entry_id":23005534,"abstract_type":"Text","abstract_text":"Growing evidence has indicated that polymorphism present in the miRNA binding site of target gene can alter the ability of miRNAs to bind its target gene and modulate the development and progression of cancer. We aimed to investigate the association between let-7 KRAS rs712 polymorphism and the risk of colorectal cancer (CRC). The let-7 KRAS rs712 was analyzed in a case-control study, including 339 CRC patients and 313 age- and sex-matched controls; the relationship between the polymorphism and the clinicopathological features of CRC was also examined. Individuals carrying the let-7 KRAS rs712 TT genotype and T allele had an increased risk of developing CRC (TT vs. GG, adjusted OR\u2009=\u20092.18; 95% CI, 1.00-4.77; T vs. G, adjusted OR\u2009=\u20091.50; 95% CI, 1.15-1.96). Stratified analyses revealed that CRC patients with the let-7 KRAS rs712 TT genotype were more likely to have clinical stage III or IV disease (OR\u2009=\u20093.29, 95% CI, 1.32-8.20) and distant metastasis (OR\u2009=\u20094.70, 95% CI, 1.81-12.25). These findings provide evidence that the let-7 KRAS rs712 polymorphism may play crucial roles in the etiology of CRC."}]},{"pmid_author":[{"pmid_author_id":7741052,"last_name":"Zhang","first_name":"Zhen","initials":"Z","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"18ac4bd06dd04dd5cdb7532b1ec1d459"},{"pmid_author_id":10784523,"last_name":"Li","first_name":"Zhao-Hui","initials":"ZH","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"d7ce1dbd016f1437242315bef5e8aee5"},{"pmid_author_id":13192794,"last_name":"Xu","first_name":"Guo-Hui","initials":"GH","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"faaaf4498ee4db8810b9f00b4f51ca49"},{"pmid_author_id":14856752,"last_name":"Gao","first_name":"Lin-Bo","initials":"LB","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8c034d9c5eb3949b6774cb771bf3c390"},{"pmid_author_id":19477763,"last_name":"Qin","first_name":"Hao-Jie","initials":"HJ","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"9de6783adcdab154fd5314c338bd5959"},{"pmid_author_id":20137637,"last_name":"Guo","first_name":"Xiao-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"bf51cfc4b6899c387c1b6bad4c651b08"},{"pmid_author_id":21341765,"last_name":"Sun","first_name":"Rui-Fen","initials":"RF","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"44e5a089880fe5e69b43a629325570ef"},{"pmid_author_id":22570289,"last_name":"Pan","first_name":"Xin-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":8897064,"orcid_id":null,"md5_hash":"82a4b3aec9f31ad760ef2293eb6234c8"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":8897064,"instit_name":"Department of Forensic Pathology, College of Forensic Medicine, Henan University of Science and Technology, Luoyang, Henan, 471003, People's Republic of China, mitochondrium@126.com.","instit_hash":"62897c6aaa12aa37497efa2331aef81f"}]}]},{"RECORD":{"pmid_entry_id":23024891,"pmid":23996697,"publication_date":"2014-03-01 00:00:00","title":"Lack of association between let-7 binding site polymorphism rs712 and risk of nasopharyngeal carcinoma.","doi":"10.1007\/s10689-013-9681-4","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":18145,"month_1910":-1250,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":18145,"journal_name":"Familial cancer","journal_abbr":"Fam Cancer","issn_print":"1389-9600","issn_online":"1573-7292","iso_abbr":"Fam Cancer","nlmid":"100898211"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":80658372,"pmid_entry_id":23024891,"pmid_author_id":14856752,"position":8},{"pmid_author_map_id":80658371,"pmid_entry_id":23024891,"pmid_author_id":13192794,"position":7},{"pmid_author_map_id":80658370,"pmid_entry_id":23024891,"pmid_author_id":19477763,"position":6},{"pmid_author_map_id":80658369,"pmid_entry_id":23024891,"pmid_author_id":7741052,"position":5},{"pmid_author_map_id":80658368,"pmid_entry_id":23024891,"pmid_author_id":10784523,"position":4},{"pmid_author_map_id":80658367,"pmid_entry_id":23024891,"pmid_author_id":20137637,"position":3},{"pmid_author_map_id":80658366,"pmid_entry_id":23024891,"pmid_author_id":7845254,"position":2},{"pmid_author_map_id":80658365,"pmid_entry_id":23024891,"pmid_author_id":22603058,"position":1}],"pmid_abstract":[{"pmid_abstract_id":21599415,"pmid_entry_id":23024891,"abstract_type":"Text","abstract_text":"Nasopharyngeal carcinoma (NPC) is characterized by its highly invasive and metastatic features. Therefore, screening genetic biomarkers of NPC to achieve early diagnose would be of great value for NPC therapy. Single nucleotide polymorphisms in let-7 miRNA binding site in 3' untranslated region of KRAS mRNA have been found to be associated with various cancer risks. In this study, we genotyped the frequency of KRAS rs712 to test its effect on NPC risk in a hospital-based case-control study in a Chinese population, with 188 histologically confirmed NPC patients and 356 cancer-free controls, using polymerase chain reaction-restriction fragment length polymorphism assay. There was no significant difference in the genotype and allele frequencies of the rs712 polymorphism between the NPC patients and the control group (GT vs. GG, OR 0.83, 95% CI 0.57-1.21; TT vs. GG, OR 1.27, 95% CI 0.58-2.75). Our data suggest that the KRAS rs712 polymorphism in let-7 miRNA binding site has no association with NPC risk. Further experiments with larger sample size or other polymorphism sites are needed to verify the result, especially in different ethnic groups."}]},{"pmid_author":[{"pmid_author_id":7741052,"last_name":"Zhang","first_name":"Zhen","initials":"Z","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"18ac4bd06dd04dd5cdb7532b1ec1d459"},{"pmid_author_id":7845254,"last_name":"Jia","first_name":"Jing","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c3fe88ec64f15b20fb4a77c6e1513715"},{"pmid_author_id":10784523,"last_name":"Li","first_name":"Zhao-Hui","initials":"ZH","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"d7ce1dbd016f1437242315bef5e8aee5"},{"pmid_author_id":13192794,"last_name":"Xu","first_name":"Guo-Hui","initials":"GH","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"faaaf4498ee4db8810b9f00b4f51ca49"},{"pmid_author_id":14856752,"last_name":"Gao","first_name":"Lin-Bo","initials":"LB","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8c034d9c5eb3949b6774cb771bf3c390"},{"pmid_author_id":19477763,"last_name":"Qin","first_name":"Hao-Jie","initials":"HJ","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"9de6783adcdab154fd5314c338bd5959"},{"pmid_author_id":20137637,"last_name":"Guo","first_name":"Xiao-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"bf51cfc4b6899c387c1b6bad4c651b08"},{"pmid_author_id":22603058,"last_name":"Pan","first_name":"Xin-Min","initials":"XM","is_valid_author":"Y","pmid_instit_id":8913417,"orcid_id":null,"md5_hash":"61bbb535b7038a9abe5906cec47b4807"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":8913417,"instit_name":"Department of Forensic Pathology, College of Forensic Medicine, Henan University of Science and Technology, Luoyang, 471003, Henan, People's Republic of China, mitochondrium@126.com.","instit_hash":"023fbacc61d1361ec28bb1352a68bf18"}]}]},{"RECORD":{"pmid_entry_id":23414217,"pmid":24416147,"publication_date":"2014-01-01 00:00:00","title":"Transcriptome-wide analysis of UTRs in non-small cell lung cancer reveals cancer-related genes with SNV-induced changes on RNA secondary structure and miRNA target sites.","doi":"10.1371\/journal.pone.0082699","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":20645,"month_1910":-1248,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":20645,"journal_name":"PloS one","journal_abbr":"PLoS One","issn_print":"","issn_online":"1932-6203","iso_abbr":"PLoS One","nlmid":"101285081"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":82630615,"pmid_entry_id":23414217,"pmid_author_id":23439175,"position":6},{"pmid_author_map_id":82630614,"pmid_entry_id":23414217,"pmid_author_id":23439174,"position":5},{"pmid_author_map_id":82630613,"pmid_entry_id":23414217,"pmid_author_id":23439173,"position":4},{"pmid_author_map_id":82630612,"pmid_entry_id":23414217,"pmid_author_id":23439172,"position":3},{"pmid_author_map_id":82630611,"pmid_entry_id":23414217,"pmid_author_id":23439171,"position":2},{"pmid_author_map_id":82630610,"pmid_entry_id":23414217,"pmid_author_id":23439170,"position":1}],"pmid_abstract":[{"pmid_abstract_id":22250966,"pmid_entry_id":23414217,"abstract_type":"Text","abstract_text":"Traditional mutation assessment methods generally focus on predicting disruptive changes in protein-coding regions rather than non-coding regulatory regions like untranslated regions (UTRs) of mRNAs. The UTRs, however, are known to have many sequence and structural motifs that can regulate translational and transcriptional efficiency and stability of mRNAs through interaction with RNA-binding proteins and other non-coding RNAs like microRNAs (miRNAs). In a recent study, transcriptomes of tumor cells harboring mutant and wild-type KRAS (V-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog) genes in patients with non-small cell lung cancer (NSCLC) have been sequenced to identify single nucleotide variations (SNVs). About 40% of the total SNVs (73,717) identified were mapped to UTRs, but omitted in the previous analysis. To meet this obvious demand for analysis of the UTRs, we designed a comprehensive pipeline to predict the effect of SNVs on two major regulatory elements, secondary structure and miRNA target sites. Out of 29,290 SNVs in 6462 genes, we predict 472 SNVs (in 408 genes) affecting local RNA secondary structure, 490 SNVs (in 447 genes) affecting miRNA target sites and 48 that do both. Together these disruptive SNVs were present in 803 different genes, out of which 188 (23.4%) were previously known to be cancer-associated. Notably, this ratio is significantly higher (one-sided Fisher's exact test p-value\u200a=\u200a0.032) than the ratio (20.8%) of known cancer-associated genes (n\u200a=\u200a1347) in our initial data set (n\u200a=\u200a6462). Network analysis shows that the genes harboring disruptive SNVs were involved in molecular mechanisms of cancer, and the signaling pathways of LPS-stimulated MAPK, IL-6, iNOS, EIF2 and mTOR. In conclusion, we have found hundreds of SNVs which are highly disruptive with respect to changes in the secondary structure and miRNA target sites within UTRs. These changes hold the potential to alter the expression of known cancer genes or genes linked to cancer-associated pathways. 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Recent studies have revealed that miRNA plays a significant role in gastric cancer development either as a tumor suppressor gene or oncogene. miRNA-single-nucleotide polymorphisms (SNPs), as a novel class of functional SNPs\/polymorphisms, have been identified as candidate biomarkers for gastric cancer susceptibility. On the basis of recent data, the present review summarizes current knowledge of the functional effects of miRNA-SNPs and their importance as candidate gastric cancer biomarkers. Additionally, this review also includes a meta-analysis of the most frequently studied miRNA-SNPs in gastric cancer. 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They have been implicated in a broad range of biological processes, and miRNA-related genetic alterations probably underlie several human diseases. Single nucleotide polymorphisms of transcripts may modulate the posttranscriptional regulation of gene expression by miRNAs and explain interindividual variability in cancer risk and in chemotherapy response. On the basis of recent association studies published in the literature, the present review mainly summarizes the potential role of miRNAs as molecular biomarkers for disease susceptibility, diagnosis, prognosis, and drug-response prediction in tumors. Many clues suggest a role for polymorphisms within the 3' untranslated regions of KRAS rs61764370, SET8 rs16917496, and MDM4 rs4245739 as SNPs in miRNA binding sites highly promising in the biology of human cancer. However, more studies are needed to better characterize the composite spectrum of genetic determinants for future use of markers in risk prediction and clinical management of diseases, heading toward personalized medicine. "}]},{"pmid_author":[{"pmid_author_id":14019969,"last_name":"Landi","first_name":"Stefano","initials":"S","is_valid_author":"Y","pmid_instit_id":4814353,"orcid_id":null,"md5_hash":"511882cee9abffb15086f00e04b94ec3"},{"pmid_author_id":22227389,"last_name":"Cipollini","first_name":"Monica","initials":"M","is_valid_author":"Y","pmid_instit_id":4814353,"orcid_id":null,"md5_hash":"18d8004fe7070edaeaa856f5094e6d3e"},{"pmid_author_id":23576577,"last_name":"Gemignani","first_name":"Federica","initials":"F","is_valid_author":"Y","pmid_instit_id":4814353,"orcid_id":null,"md5_hash":"86625c735006ec02930828f69d27d362"}]},{"pmid_instit":[{"pmid_instit_id":4814353,"instit_name":"Department of Biology, University of Pisa, Pisa, Italy.","instit_hash":"54bcf24d0a38b2c64271888c8115e947"}]}]},{"RECORD":{"pmid_entry_id":24144719,"pmid":25201577,"publication_date":"2014-10-01 00:00:00","title":"Association between a functional polymorphism rs712 within let-7-binding site and risk of papillary thyroid cancer.","doi":"10.1007\/s12032-014-0221-3","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":7493,"month_1910":-1257,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":7493,"journal_name":"Medical oncology (Northwood, London, England)","journal_abbr":"Med Oncol","issn_print":"1357-0560","issn_online":"1559-131X","iso_abbr":"Med Oncol","nlmid":"9435512"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":86424229,"pmid_entry_id":24144719,"pmid_author_id":7748738,"position":9},{"pmid_author_map_id":86424228,"pmid_entry_id":24144719,"pmid_author_id":15996858,"position":8},{"pmid_author_map_id":86424227,"pmid_entry_id":24144719,"pmid_author_id":25804001,"position":7},{"pmid_author_map_id":86424226,"pmid_entry_id":24144719,"pmid_author_id":8298153,"position":6},{"pmid_author_map_id":86424225,"pmid_entry_id":24144719,"pmid_author_id":23187708,"position":5},{"pmid_author_map_id":86424224,"pmid_entry_id":24144719,"pmid_author_id":21995599,"position":4},{"pmid_author_map_id":86424223,"pmid_entry_id":24144719,"pmid_author_id":25804000,"position":3},{"pmid_author_map_id":86424222,"pmid_entry_id":24144719,"pmid_author_id":23187709,"position":2},{"pmid_author_map_id":86424221,"pmid_entry_id":24144719,"pmid_author_id":25803999,"position":1}],"pmid_abstract":[{"pmid_abstract_id":23489774,"pmid_entry_id":24144719,"abstract_type":"Text","abstract_text":"KRAS mutation is frequently detected in a series of cancers, including papillary thyroid cancer (PTC). Recently, a genetic variant of rs712 in the 3' untranslated region of the KRAS gene has been reported to be functional in the regulation of KRAS by disrupting complementary site of let-7 and miR-181. We aimed to investigate whether the polymorphism is a risk factor for PTC. We conducted an association study, including 252 PTC patients and 290 healthy controls. The KRAS rs712 polymorphism was genotyped by polymerase chain reaction-restriction fragment length polymorphism. Although no significant difference of the KRAS rs712 distribution was observed between cases and controls in overall analysis, stratification analysis showed that patients carrying the KRAS rs712TT genotype were less likely to develop stages T3 and T4 under a recessive genetic model (OR 0.26, 95% CI 0.08-0.82). These results supported the role of the KRAS rs712 polymorphism as a potential genetic biomarker for the extension of PTC. Further population-based association studies are of great value to confirm the results in diverse ethnicities."}]},{"pmid_author":[{"pmid_author_id":7748738,"last_name":"Zhang","first_name":"Lin","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0687ca809b6714279382fe0456f35864"},{"pmid_author_id":8298153,"last_name":"Chen","first_name":"Peng","initials":"P","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1c8247e3b3050f86102e96bc35c85ae1"},{"pmid_author_id":15996858,"last_name":"Gao","first_name":"Linbo","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"25fe26cb32284c3dd1f62a77ff9d4df2"},{"pmid_author_id":21995599,"last_name":"Zhu","first_name":"Jingqiang","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"5906ba4c0588470e387995c3fcfc014b"},{"pmid_author_id":23187708,"last_name":"Sun","first_name":"Ruifen","initials":"R","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1ffb9a3655c98eeff60e08fa43f2d29d"},{"pmid_author_id":23187709,"last_name":"Liang","first_name":"Yundan","initials":"Y","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"6a7331cb28e3ed5a4f499f65f9b4f4cf"},{"pmid_author_id":25803999,"last_name":"Jin","first_name":"Hong","initials":"H","is_valid_author":"Y","pmid_instit_id":10460411,"orcid_id":null,"md5_hash":"4dde16b76ea0182abce76eb182e3a6e2"},{"pmid_author_id":25804000,"last_name":"Wang","first_name":"Xunli","initials":"X","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c992a1f276f8680bba1a6c472820d3d6"},{"pmid_author_id":25804001,"last_name":"Nie","first_name":"Xinwen","initials":"X","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"6ab519ab1e56290e0ec9cbd73f61330e"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":10460411,"instit_name":"Department of Immunology, West China School of Preclinical and Forensic Medicine, Sichuan University, Chengdu, 610041, Sichuan, People's Republic of China.","instit_hash":"abab929e4f5fb08a2804faf79bbf40fd"}]}]},{"RECORD":{"pmid_entry_id":25064270,"pmid":26178671,"publication_date":"2015-01-01 00:00:00","title":"Let-7 related genetic variation and risk of metabolic syndrome in a Chinese population.","doi":"10.1507\/endocrj.EJ15-0236","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":7471,"month_1910":-1260,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":7471,"journal_name":"Endocrine journal","journal_abbr":"Endocr J","issn_print":"0918-8959","issn_online":"1348-4540","iso_abbr":"Endocr J","nlmid":"9313485"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":91558019,"pmid_entry_id":25064270,"pmid_author_id":8074799,"position":7},{"pmid_author_map_id":91558018,"pmid_entry_id":25064270,"pmid_author_id":7830811,"position":6},{"pmid_author_map_id":91558017,"pmid_entry_id":25064270,"pmid_author_id":8044899,"position":4},{"pmid_author_map_id":91558016,"pmid_entry_id":25064270,"pmid_author_id":7733066,"position":3},{"pmid_author_map_id":91558015,"pmid_entry_id":25064270,"pmid_author_id":14020123,"position":2},{"pmid_author_map_id":91558014,"pmid_entry_id":25064270,"pmid_author_id":22439065,"position":1},{"pmid_author_map_id":91555065,"pmid_entry_id":25064270,"pmid_author_id":7674354,"position":5}],"pmid_abstract":[{"pmid_abstract_id":25097651,"pmid_entry_id":25064270,"abstract_type":"Text","abstract_text":"MicroRNA (miRNA) related genetic variation may change miRNA binding affinity and mRNA expression levels of the target genes, thus leading to altered metabolic parameters. This study explored the influence of let-7 related single nucleotide polymorphisms (SNPs) on individual susceptibility to metabolic syndrome (MetS) in a Chinese population. Seven SNPs located at the pri-let-7 gene region, pre-let-7 gene region or 3'-UTR of the KRAS gene were selected. The SNPs were genotyped in 503 MetS patients and 529 normal controls using the high-throughput Sequenom genotyping platform. Unconditional logistic regression analysis was utilized to estimate the association between these SNPs and the risk of MetS. There are three SNPs significantly associated with MetS. The A allele of rs17276588 was associated with an increased risk effect for MetS (Adjusted OR=1.75, 95%CI 1.37-2.25, P<0.001). Rs10993081 AG genotype was significantly associated with an increased risk of MetS compared with AA genotypes (Adjusted OR=1.42, 95%CI 1.11-1.83, P=0.006). Rs10877887 TC genotype was significantly associated with an increased risk of MetS compared with TT genotypes (Adjusted OR=1.52, 95% CI 1.16-1.99, P=0.002). Additionally, interactions between rs7045890 and rs712, rs17276588 and rs10877887 were significantly associated with risk of MetS. In conclusion, our study found that let-7 related genetic variation is associated with MetS and may contribute to the susceptibility of MetS. Larger, prospective studies are warranted to validate our findings. "}]},{"pmid_author":[{"pmid_author_id":7674354,"last_name":"Wang","first_name":"Wei","initials":"W","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"bbda9e10963849b79f5d1f4c3efd7455"},{"pmid_author_id":7733066,"last_name":"Zhang","first_name":"Jie","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"395c5714abb0f748eb2ec30865f0fba8"},{"pmid_author_id":7830811,"last_name":"Dong","first_name":"Jing","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"cc7a2c1a338d6ead0ed4ea58a2620672"},{"pmid_author_id":8044899,"last_name":"Wang","first_name":"Shuo","initials":"S","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8d77827136e722c558962d6bcb6048dd"},{"pmid_author_id":8074799,"last_name":"He","first_name":"Yan","initials":"Y","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"e5fb1f851114cc11f761fe8c7a4f6dbc"},{"pmid_author_id":14020123,"last_name":"Wu","first_name":"Li-Juan","initials":"LJ","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"01236fbf690e987ed54ceb94d0c2a06e"},{"pmid_author_id":22439065,"last_name":"Yan","first_name":"Yu-Xiang","initials":"YX","is_valid_author":"Y","pmid_instit_id":8833548,"orcid_id":null,"md5_hash":"3bf473cc0e49723310176346459d1deb"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":8833548,"instit_name":"Department of Epidemiology and Biostatistics, School of Public Health, Capital Medical University, Beijing, China.","instit_hash":"4514990b88cadba03cf9eac0181ea601"}]}]},{"RECORD":{"pmid_entry_id":25134981,"pmid":26252302,"publication_date":"2015-08-01 00:00:00","title":"A Functional Polymorphism in the Promoter of MiR-143\/145 Is Associated With the Risk of Cervical Squamous Cell Carcinoma in Chinese Women: A Case-Control Study.","doi":"10.1097\/MD.0000000000001289","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":468,"month_1910":-1267,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":468,"journal_name":"Medicine","journal_abbr":"Medicine (Madr)","issn_print":"0304-5412","issn_online":"1578-8822","iso_abbr":"Medicine (Madr)","nlmid":"101470745"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":91972919,"pmid_entry_id":25134981,"pmid_author_id":15996858,"position":10},{"pmid_author_map_id":91972918,"pmid_entry_id":25134981,"pmid_author_id":7748738,"position":9},{"pmid_author_map_id":91972916,"pmid_entry_id":25134981,"pmid_author_id":8298153,"position":8},{"pmid_author_map_id":91972915,"pmid_entry_id":25134981,"pmid_author_id":25804001,"position":7},{"pmid_author_map_id":91972914,"pmid_entry_id":25134981,"pmid_author_id":9137979,"position":5},{"pmid_author_map_id":91972913,"pmid_entry_id":25134981,"pmid_author_id":8825917,"position":4},{"pmid_author_map_id":91972912,"pmid_entry_id":25134981,"pmid_author_id":10487808,"position":3},{"pmid_author_map_id":91972911,"pmid_entry_id":25134981,"pmid_author_id":23187708,"position":2},{"pmid_author_map_id":91972910,"pmid_entry_id":25134981,"pmid_author_id":29509207,"position":1},{"pmid_author_map_id":91972800,"pmid_entry_id":25134981,"pmid_author_id":7830563,"position":6}],"pmid_abstract":[{"pmid_abstract_id":25220665,"pmid_entry_id":25134981,"abstract_type":"Text","abstract_text":"MiR-143\/145 is down-regulated in cervical cancer, which may serve as a tumor suppressor by targeting KRAS and Ras-responsive element-binding protein (RREB1). Activated KRAS leads to down-regulation of miR-143\/145 transcription in a RREB1-dependent manner, establishing a miR-143\/145-KRAS-RREB1 feedback loop. A polymorphism rs4705343C\/T in the promoter of miR-143\/145 might influence the binding of TATA-binding protein. We hypothesized that the miR-143\/145 rs4705343 and KRAS rs712 may be related to the occurrence of cervical squamous cell carcinoma (CSCC). In this study, we genotyped the 2 polymorphisms in 415 patients with CSCC and 504 controls using polymerase chain reaction-restriction fragment length polymorphism. The promoter activities were measured by the Dual-Luciferase Reporter Assay System. We found that the rs4705343TC genotype was associated with an increased risk of CSCC (adjusted odds ratio [OR]\u200a=\u200a1.37; 95% confidence interval [CI], 1.05-1.80). The significantly increased association was also observed in a dominant genetic model (adjusted OR\u200a=\u200a1.32; 95% CI, 1.01-1.72). Combined analysis showed that individuals carrying the genotypes of rs4705343 TC\/CC and rs712GT\/TT had a 1.47-fold increased risk of CSCC (adjusted OR\u200a=\u200a1.47; 95% CI, 1.01-2.15). By using multifactor dimensionality reduction software method, we identified a significant interaction between the miR-143\/145 rs4705343 and KRAS rs712. Dual-Luciferase Reporter Assay showed that the luciferase activity was significantly lower in cells transfected with the rs4705343C allele than that of the rs4705343T allele. These findings indicate that miR-143\/145 rs4705343 and KRAS rs712 may contribute to the etiology of CSCC in Chinese women."}]},{"pmid_author":[{"pmid_author_id":7748738,"last_name":"Zhang","first_name":"Lin","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0687ca809b6714279382fe0456f35864"},{"pmid_author_id":7830563,"last_name":"Wang","first_name":"Li","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"58b8b8efb4494de912fc4164354307eb"},{"pmid_author_id":8298153,"last_name":"Chen","first_name":"Peng","initials":"P","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1c8247e3b3050f86102e96bc35c85ae1"},{"pmid_author_id":8825917,"last_name":"Yuan","first_name":"Fang","initials":"F","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1faf7b553749feacdf813a122e117679"},{"pmid_author_id":9137979,"last_name":"Liang","first_name":"Weibo","initials":"W","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1d38d10de4cf80483a8146fa0a0e3f6f"},{"pmid_author_id":10487808,"last_name":"Li","first_name":"Lijuan","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"867d6fa1ca5ffe00a249d65c1f613dc0"},{"pmid_author_id":15996858,"last_name":"Gao","first_name":"Linbo","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"25fe26cb32284c3dd1f62a77ff9d4df2"},{"pmid_author_id":23187708,"last_name":"Sun","first_name":"Ruifen","initials":"R","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1ffb9a3655c98eeff60e08fa43f2d29d"},{"pmid_author_id":25804001,"last_name":"Nie","first_name":"Xinwen","initials":"X","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"6ab519ab1e56290e0ec9cbd73f61330e"},{"pmid_author_id":29509207,"last_name":"Liang","first_name":"Yundan","initials":"Y","is_valid_author":"Y","pmid_instit_id":12219824,"orcid_id":null,"md5_hash":"365be5949150eac12e76867faa1b08a6"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":12219824,"instit_name":"From the Laboratory of Molecular and Translational Medicine, West China Institute of Women and Children's Health, West China Second University Hospital, Sichuan University, Chengdu, Sichuan, P.R. China (YL, RS, LL, FY, XN, LZ, LG); Key Laboratory of Obstetric and Gynecologic and Pediatric Diseases and Birth Defects of Ministry of Education, Chengdu, Sichuan, P.R. China (YL, RS, LL, FY, XN, LZ, LG); Central Laboratory, Yunnan University of Chinese Traditional Medicine, Kunming, Yunnan, P.R. China (RS); and Department of Forensic Biology, West China School of Preclinical and Forensic Medicine, Sichuan University, Chengdu, Sichuan, P.R. China (LW, PC, LZ).","instit_hash":"1c59ef5cdf5d98a4af8259b897a71d7d"}]}]},{"RECORD":{"pmid_entry_id":25197173,"pmid":26316738,"publication_date":"2015-01-01 00:00:00","title":"A variant in 3'-untranslated region of KRAS compromises its interaction with hsa-let-7g and contributes to the development of lung cancer in patients with COPD.","doi":"10.2147\/COPD.S83596","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":20730,"month_1910":-1260,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":20730,"journal_name":"International journal of chronic obstructive pulmonary disease","journal_abbr":"Int J Chron Obstruct Pulmon Dis","issn_print":"1176-9106","issn_online":"1178-2005","iso_abbr":"Int J Chron Obstruct Pulmon Dis","nlmid":"101273481"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":92311705,"pmid_entry_id":25197173,"pmid_author_id":29762096,"position":5},{"pmid_author_map_id":92311704,"pmid_entry_id":25197173,"pmid_author_id":29762095,"position":4},{"pmid_author_map_id":92311703,"pmid_entry_id":25197173,"pmid_author_id":29762094,"position":3},{"pmid_author_map_id":92311702,"pmid_entry_id":25197173,"pmid_author_id":29762093,"position":2},{"pmid_author_map_id":92311701,"pmid_entry_id":25197173,"pmid_author_id":29762092,"position":1}],"pmid_abstract":[{"pmid_abstract_id":25328197,"pmid_entry_id":25197173,"abstract_type":"OBJECTIVE","abstract_text":"The objective of the present study was to explore the molecular mechanism by which a single nucleotide polymorphism (rs712) interferes with interaction between 3'-untranslated region (3'-UTR) of KRAS and let-7g, and its association with development of lung cancer in the patients with COPD."},{"pmid_abstract_id":25328198,"pmid_entry_id":25197173,"abstract_type":"MATERIALS AND METHODS","abstract_text":"In this study, we confirmed that KRAS is a target of let-7g in lung cancer cells, and that introduction of rs712 minor allele into 3'-UTR significantly compromised the miRNA\/mRNA interaction by using a luciferase reporter system. Additionally, a total of 35 lung tissue samples were obtained (TT:17, TG:12, GG:6), and let-7g and KRAS expression levels were determined."},{"pmid_abstract_id":25328199,"pmid_entry_id":25197173,"abstract_type":"RESULTS","abstract_text":"We showed that let-7g level was similar between groups, and the concentration of KRAS in GG genotype group was significantly higher than in TT or GT genotype group. Meanwhile, we found COPD patients with GG genotype had significantly higher risk for lung cancer (odds ratio OR =6.83, P=0.0081), compared with TT and GT genotypes."},{"pmid_abstract_id":25328200,"pmid_entry_id":25197173,"abstract_type":"CONCLUSION","abstract_text":"Our study demonstrated that KRAS 3'-UTR rs712 polymorphism interfered with miRNA\/mRNA interaction, and showed that the minor allele was associated with an elevated risk for development of lung cancer in COPD."}]},{"pmid_author":[{"pmid_author_id":29762092,"last_name":"Hu","first_name":"Hua","initials":"H","is_valid_author":"Y","pmid_instit_id":12338419,"orcid_id":null,"md5_hash":"1082f6595c139d31939c26f4141afc69"},{"pmid_author_id":29762093,"last_name":"Zhang","first_name":"Linlin","initials":"L","is_valid_author":"Y","pmid_instit_id":12338419,"orcid_id":null,"md5_hash":"ef34bdf144bc8aef61fa24b9f4944b58"},{"pmid_author_id":29762094,"last_name":"Teng","first_name":"Geling","initials":"G","is_valid_author":"Y","pmid_instit_id":12338419,"orcid_id":null,"md5_hash":"a43130d5791bb6e7ac3dc7f8db345aef"},{"pmid_author_id":29762095,"last_name":"Wu","first_name":"Yanhua","initials":"Y","is_valid_author":"Y","pmid_instit_id":12338419,"orcid_id":null,"md5_hash":"dd13988315f0d862cf160fbcc0e6a7ee"},{"pmid_author_id":29762096,"last_name":"Chen","first_name":"Ying","initials":"Y","is_valid_author":"Y","pmid_instit_id":12338420,"orcid_id":null,"md5_hash":"e0895fa3138ab57f34403518b8ca6aee"}]},{"pmid_instit":[{"pmid_instit_id":12338419,"instit_name":"The Respiratory Department, Shandong Provincial Chest Hospital, Jinan, Shandong, People's Republic of China.","instit_hash":"04e9670b1f4ba958106f65275623fd37"},{"pmid_instit_id":12338420,"instit_name":"The Clinical Laboratory of Shandong Provincial Chest Hospital, Jinan, Shandong, People's Republic of China.","instit_hash":"a7321e9512e5c5d3172731d8f615287c"}]}]},{"RECORD":{"pmid_entry_id":25403684,"pmid":26535719,"publication_date":"2015-10-30 00:00:00","title":"An let-7 KRAS rs712 polymorphism increases hepatocellular carcinoma risk.","doi":"10.4238\/2015.October.29.24","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":18164,"month_1910":-1269,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":18164,"journal_name":"Genetics and molecular research : GMR","journal_abbr":"Genet Mol Res","issn_print":"","issn_online":"1676-5680","iso_abbr":"Genet Mol Res","nlmid":"101169387"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":93440618,"pmid_entry_id":25403684,"pmid_author_id":30599809,"position":4},{"pmid_author_map_id":93440617,"pmid_entry_id":25403684,"pmid_author_id":30599808,"position":3},{"pmid_author_map_id":93440616,"pmid_entry_id":25403684,"pmid_author_id":30599807,"position":2},{"pmid_author_map_id":93440615,"pmid_entry_id":25403684,"pmid_author_id":30599806,"position":1}],"pmid_abstract":[{"pmid_abstract_id":25685646,"pmid_entry_id":25403684,"abstract_type":"Text","abstract_text":"KRAS, also known as V-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog, acts as an intracellular signal transducer. The oncogenic KRAS mutation is an essential step in the development of many types of human cancers, including hepatocellular carcinoma. Here we aimed to investigate the relationship between KRAS rs712 polymorphisms and hepatocellular carcinoma susceptibility. Five-hundred-and-fourteen participants were enrolled in a case-control study (262 cases and 252 normal subjects). The variants were distinguished using polymerase chain reaction-restriction fragment length polymorphism. Significantly increased HCC risk was observed to be associated with the T allele of the rs712 locus (P = 0.049, OR = 1.35, 95%CI = 1.01-1.78). Further, HCC risk with the GT genotype (P = 0.015, OR = 1.64, 95%CI = 1.08-2.50) and the TT genotype (P = 0.015, OR = 2.56, 95%CI = 1.05-6.25) in a codominant model was significantly higher than that with the GG genotype. In a dominant model, significantly increased HCC susceptibility was also associated with T allele carriers (P = 0.006, OR = 1.75, 95%CI = 1.16-2.63). Moreover, we found that the frequency of the KRAS rs712 TT genotype was significantly higher in HBV-positive HCC patients than in HBV-negative HCC patients."}]},{"pmid_author":[{"pmid_author_id":30599806,"last_name":"Xiong","first_name":"D","initials":"D","is_valid_author":"Y","pmid_instit_id":12730643,"orcid_id":null,"md5_hash":"8a2725bcbaead8d96d0fe4ae2b589c96"},{"pmid_author_id":30599807,"last_name":"Song","first_name":"Y P","initials":"YP","is_valid_author":"Y","pmid_instit_id":12730644,"orcid_id":null,"md5_hash":"d0db9054f84f5d94a4c19d911b693cec"},{"pmid_author_id":30599808,"last_name":"Xiong","first_name":"W","initials":"W","is_valid_author":"Y","pmid_instit_id":12730645,"orcid_id":null,"md5_hash":"f61923a8ea6deea8957d89c1a21d3c94"},{"pmid_author_id":30599809,"last_name":"Liang","first_name":"Y D","initials":"YD","is_valid_author":"Y","pmid_instit_id":12730644,"orcid_id":null,"md5_hash":"82e409c4df9df5c629bc502d8e23e31a"}]},{"pmid_instit":[{"pmid_instit_id":12730643,"instit_name":"Department of General Surgery, Meishan People's Hospital, Meishan, Sichuan, China.","instit_hash":"5104d763c7abc876bb1ca30522c69cd9"},{"pmid_instit_id":12730644,"instit_name":"Laboratory of Molecular Translational Medicine, West China Institute of Women and Children's Health.","instit_hash":"4dcf5d6f429c0de1f5425e2606e03fa6"},{"pmid_instit_id":12730645,"instit_name":"Multiple Organ Transplant Center, Sichuan Provincial People's Hospital, Chengdu, China.","instit_hash":"9dde206889d1732471e986d6b65e9ea4"}]}]},{"RECORD":{"pmid_entry_id":25797453,"pmid":26959740,"publication_date":"2016-04-05 00:00:00","title":"A single-nucleotide polymorphism in the 3'-UTR region of the adipocyte fatty acid binding protein 4 gene is associated with prognosis of triple-negative breast cancer.","doi":"10.18632\/oncotarget.7920","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":25131,"month_1910":-1275,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":25131,"journal_name":"Oncotarget","journal_abbr":"Oncotarget","issn_print":"","issn_online":"1949-2553","iso_abbr":"Oncotarget","nlmid":"101532965"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":95611249,"pmid_entry_id":25797453,"pmid_author_id":32192870,"position":9},{"pmid_author_map_id":95611248,"pmid_entry_id":25797453,"pmid_author_id":32192869,"position":8},{"pmid_author_map_id":95611247,"pmid_entry_id":25797453,"pmid_author_id":32192868,"position":7},{"pmid_author_map_id":95611246,"pmid_entry_id":25797453,"pmid_author_id":32192867,"position":6},{"pmid_author_map_id":95611245,"pmid_entry_id":25797453,"pmid_author_id":32192866,"position":5},{"pmid_author_map_id":95611244,"pmid_entry_id":25797453,"pmid_author_id":32192865,"position":4},{"pmid_author_map_id":95611243,"pmid_entry_id":25797453,"pmid_author_id":32192864,"position":3},{"pmid_author_map_id":95611242,"pmid_entry_id":25797453,"pmid_author_id":32192863,"position":2},{"pmid_author_map_id":95611241,"pmid_entry_id":25797453,"pmid_author_id":27414818,"position":1}],"pmid_abstract":[{"pmid_abstract_id":26369143,"pmid_entry_id":25797453,"abstract_type":"Text","abstract_text":"Triple-negative breast cancer (TNBC) is a subtype of breast cancer with poor prognosis and high heterogeneity. The aim of this study was to screen patients for single-nucleotide polymorphisms (SNPs) associated with the prognosis of TNBC. Database-derived SNPs (NextBio, Ensembl, NCBI and MirSNP) located in the 3'-untranslated regions (3'-UTRs) of genes that are differentially expressed in breast cancer were selected. The possible associations between 111 SNPs and progression risk among 323 TNBC patients were investigated using a two-step case-control study with a discovery cohort (n=162) and a validation cohort (n=161). We identified the rs1054135 SNP in the adipocyte fatty acid binding protein 4 (FABP4) gene as a predictor of TNBC recurrence. The G allele of rs1054135 was associated with a reduced risk of disease progression as well as a prolonged disease-free survival time (DFS), with a hazard ratio (HR) for recurrence in the combined sample of 0.269 [95%CI: 0.098-0.735;P=0.001]. Notably, for individuals having the rs1054135 SNP with the AA\/AG genotype, the magnitude of increased tumour recurrence risk for overweight patients (BMI\u226525kg\/m2) was significantly elevated (HR2.53; 95%CI: 1.06-6.03). Immunohistochemical staining of adipocytes adjacent to TNBC tissues showed that the expression level of FABP4 was statistically significantly lower in patients with the rs1054135-GG genotype and those in the disease-free group (P=0.0004 and P=0.0091, respectively). These results suggested that the expression of a lipid metabolism-related gene and an important SNP in the 3'-UTR of FABP4 are associated with TNBC prognosis, which may aid in the screening of high-risk patients with TNBC recurrence and the development of novel chemotherapeutic agents. "}]},{"pmid_author":[{"pmid_author_id":27414818,"last_name":"Wang","first_name":"Wenmiao","initials":"W","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"1343eea59a9c20fdf9d73df56a7d7f40"},{"pmid_author_id":32192863,"last_name":"Yuan","first_name":"Peng","initials":"P","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"513bc8b25379009f5aaacbf777f4b6d1"},{"pmid_author_id":32192864,"last_name":"Yu","first_name":"Dianke","initials":"D","is_valid_author":"Y","pmid_instit_id":13465287,"orcid_id":null,"md5_hash":"7c4a3884120e86bc03c4b5a67a4b7732"},{"pmid_author_id":32192865,"last_name":"Du","first_name":"Feng","initials":"F","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"7c1ec32e44f45bcee3e965e4b759981b"},{"pmid_author_id":32192866,"last_name":"Zhu","first_name":"Anjie","initials":"A","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"ab8ccf1bb4e6a7db97197d1c70b77873"},{"pmid_author_id":32192867,"last_name":"Li","first_name":"Qing","initials":"Q","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"5cd8d13c9620d9a78f59f7ebc7c9ec21"},{"pmid_author_id":32192868,"last_name":"Zhang","first_name":"Pin","initials":"P","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"ba51ac3ac103806d13b8d936c5199331"},{"pmid_author_id":32192869,"last_name":"Lin","first_name":"Dongxin","initials":"D","is_valid_author":"Y","pmid_instit_id":13465287,"orcid_id":null,"md5_hash":"abd0421d7dd37222fa0db45bdda493fd"},{"pmid_author_id":32192870,"last_name":"Xu","first_name":"Binghe","initials":"B","is_valid_author":"Y","pmid_instit_id":13465286,"orcid_id":null,"md5_hash":"cb0c5e6c0f18f5a30073a668f776c9f6"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":13465286,"instit_name":"Department of Medical Oncology, Cancer Institute and Hospital, Peking Union Medical College, Chinese Academy of Medical Sciences, Beijing, P.R. China.","instit_hash":"0f79f983b6ed0614e0111c58bf2c8d8c"},{"pmid_instit_id":13465287,"instit_name":"Department of Etiology and Carcinogenesis, Cancer Institute and Hospital, Chinese Academy of Medical Science and Peking Union Medical College, Beijing, P.R. China.","instit_hash":"31add3a4796a3b898068b0a7c75568a4"}]}]},{"RECORD":{"pmid_entry_id":26782569,"pmid":28099923,"publication_date":"2017-02-21 00:00:00","title":"Significant association between Let-7-KRAS rs712 G > T polymorphism and cancer risk in the Chinese population: a meta-analysis.","doi":"10.18632\/oncotarget.14672","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":25131,"month_1910":-1285,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":25131,"journal_name":"Oncotarget","journal_abbr":"Oncotarget","issn_print":"","issn_online":"1949-2553","iso_abbr":"Oncotarget","nlmid":"101532965"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":100953219,"pmid_entry_id":26782569,"pmid_author_id":8141800,"position":8},{"pmid_author_map_id":100953218,"pmid_entry_id":26782569,"pmid_author_id":18800053,"position":7},{"pmid_author_map_id":100953217,"pmid_entry_id":26782569,"pmid_author_id":36166201,"position":6},{"pmid_author_map_id":100953216,"pmid_entry_id":26782569,"pmid_author_id":36166200,"position":5},{"pmid_author_map_id":100953215,"pmid_entry_id":26782569,"pmid_author_id":36166199,"position":4},{"pmid_author_map_id":100953214,"pmid_entry_id":26782569,"pmid_author_id":36166198,"position":3},{"pmid_author_map_id":100953213,"pmid_entry_id":26782569,"pmid_author_id":31168808,"position":2},{"pmid_author_map_id":100953212,"pmid_entry_id":26782569,"pmid_author_id":33630402,"position":1}],"pmid_abstract":[{"pmid_abstract_id":28054231,"pmid_entry_id":26782569,"abstract_type":"Text","abstract_text":"Association between let-7-KRAS rs712 polymorphism and cancer risk was inconsistent. We therefore conducted this meta-analysis to clarify the association between let-7-KRAS rs712 polymorphism and cancer risk with STATA 14.0 software. A systemic literature search in online databases (PubMed, Embase, CNKI and Wanfang database) was preformed to obtain relevant articles. A total of 13 case-control studies involving 3,453 patients and 4,470 controls were identified up to May 16, 2015. The pooled results indicated that significantly increased risk were observed in Chinese population in T vs. G (OR = 1.21, 95% CI = 1.03-1.42) and TT vs. GG + GT genetic models (OR = 1.69, 95% CI = 1.17-2.42). Sensitivity analysis was conducted and the result without heterogeneity showed significant associations in all five genetic models. Subgroup analyses of cancer type indicated a similar result in digestive cancer (for T vs. G: OR = 1.41, 95% CI = 1.26-1.57; GT vs. GG: OR = 1.24, 95% CI = 1.07-1.43; TT vs. GG: OR = 2.53, 95% CI = 1.86-3.44; GT + TT vs. GG: OR = 1.36, 95% CI = 1.19-1.56; TT vs. GG + GT: OR = 2.35, 95% CI = 1.73-3.19). In summary, these evidences demonstrate that let-7-KRAS rs712 G > T polymorphism might be associated with digestive system cancer risk in the Chinese population."}]},{"pmid_author":[{"pmid_author_id":8141800,"last_name":"Shen","first_name":"Ming","initials":"M","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"ef3b432125b3543c8475880bffa4d965"},{"pmid_author_id":18800053,"last_name":"Niu","first_name":"Yu-Ming","initials":"YM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c1be3ca3b48e038a0d1dfdc09cd197be"},{"pmid_author_id":31168808,"last_name":"Hu","first_name":"Yuan-Yuan","initials":"YY","is_valid_author":"Y","pmid_instit_id":10710318,"orcid_id":null,"md5_hash":"efd426dba8b915fce5bf0959fab460ec"},{"pmid_author_id":33630402,"last_name":"Du","first_name":"Xin-Ya","initials":"XY","is_valid_author":"Y","pmid_instit_id":14111179,"orcid_id":null,"md5_hash":"9b65889a635e1d52be7d4c7400ce762d"},{"pmid_author_id":36166198,"last_name":"Xie","first_name":"Chun","initials":"C","is_valid_author":"Y","pmid_instit_id":14111179,"orcid_id":null,"md5_hash":"4bd1afa4359bdb303e39bcb1feab31b3"},{"pmid_author_id":36166199,"last_name":"Deng","first_name":"Chun-Yan","initials":"CY","is_valid_author":"Y","pmid_instit_id":15243864,"orcid_id":null,"md5_hash":"9809b8ed635f33ecad0ba8f2a69d9a3d"},{"pmid_author_id":36166200,"last_name":"Liu","first_name":"Cai-Yun","initials":"CY","is_valid_author":"Y","pmid_instit_id":10710318,"orcid_id":null,"md5_hash":"987971c2d37790d41636ac5eaa1e5c0d"},{"pmid_author_id":36166201,"last_name":"Luo","first_name":"Zhi-Guo","initials":"ZG","is_valid_author":"Y","pmid_instit_id":15243865,"orcid_id":null,"md5_hash":"e194f29c85c3b5d7c89e233839a35d6c"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":10710318,"instit_name":"Department of Stomatology, Taihe Hospital, Hubei University of Medicine, Shiyan 442000, China.","instit_hash":"852b513399cd1f632076b3aaf7f7be84"},{"pmid_instit_id":14111179,"instit_name":"Department of Stomatology, People's Hospital of New District Longhua Shenzhen, Shenzhen 518109, China.","instit_hash":"6792067a73e122b702560fec2d3fd1ab"},{"pmid_instit_id":15243864,"instit_name":"Intensive Care Unit, Taihe Hospital, Hubei University of Medicine, Shiyan 442000, China.","instit_hash":"d2ce31a30562a2832932bae2ed4a224f"},{"pmid_instit_id":15243865,"instit_name":"Department of Clinical Oncology, Taihe Hospital, Hubei University of Medicine, Shiyan 442000, China.","instit_hash":"0fee67bbfcfd15c6de12e0312d158ae9"}]}]},{"RECORD":{"pmid_entry_id":27260019,"pmid":28610418,"publication_date":"1969-12-31 00:00:00","title":"KRAS Gene Polymorphisms and their Impact on Breast Cancer Risk in an Iranian Population","doi":"NULL","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":18075,"month_1910":-719,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":18075,"journal_name":"Asian Pacific journal of cancer prevention : APJCP","journal_abbr":"Asian Pac J Cancer Prev","issn_print":"1513-7368","issn_online":"2476-762X","iso_abbr":"Asian Pac J Cancer Prev","nlmid":"101130625"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":103730969,"pmid_entry_id":27260019,"pmid_author_id":21347162,"position":5},{"pmid_author_map_id":103730968,"pmid_entry_id":27260019,"pmid_author_id":25172043,"position":4},{"pmid_author_map_id":103730967,"pmid_entry_id":27260019,"pmid_author_id":38116972,"position":3},{"pmid_author_map_id":103730966,"pmid_entry_id":27260019,"pmid_author_id":10945674,"position":2},{"pmid_author_map_id":103730965,"pmid_entry_id":27260019,"pmid_author_id":38116971,"position":1}],"pmid_abstract":[{"pmid_abstract_id":28868395,"pmid_entry_id":27260019,"abstract_type":"Text","abstract_text":"Single nucleotide polymorphisms (SNPs) in the let-7 miRNA binding site within the 3\u2019 untranslated region (3\u2019UTR)\nof KRAS appear related to the risk of cancer. The present case-control study was conducted with 244 BC patients and 204\nhealthy women to examine whether KRAS polymorphisms (rs61764370 T\/G and rs712 G\/T) are associated with breast\ncancer (BC) risk in an Iranian population. The polymerase chain reaction restriction fragment length polymorphism\n(PCR-RFLP) method was used for genotyping of KRAS SNPs. Our results showed that the rs61764370 TG genotype\n(OR= 3.73; 95% CI =1.38-10.08; P=0.007) as well as the G allele OR= 3.56; 95% CI =1.33-9.53; P=0.008, respectively)\nincreased the risk of BC. However, the KRAS rs712 TT vs GG+GT genotype in a recessive model was associated with\na reduced risk of BC (OR= 0.56; 95% CI =0.38-0.84; P=0.006). In addition, the rs712 T allele decreased the risk of\nBC compared with the G allele (OR=0.75, 95%CI=0.58-0.97, P=0.031). However, we found no relationship among\nKRAS SNPs and clinicopathological characteristics of BC patients (P>0.05). Taken together, the present study provided\nevidence of relationships between KRAS polymorphisms and BC risk in a southeast Iranian population. Additional\nstudies using larger sample sizes and diverse ethnicities are now warranted."}]},{"pmid_author":[{"pmid_author_id":10945674,"last_name":"Hashemi","first_name":"Mohammad","initials":"M","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"a11d25c3276ef59ff832d54cbf6cd9af"},{"pmid_author_id":21347162,"last_name":"Bahari","first_name":"Gholamreza","initials":"G","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8c80bda6addaa47f0c926b9b236d975b"},{"pmid_author_id":25172043,"last_name":"Hashemi","first_name":"Seyed Mehdi","initials":"SM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"dac3deac2da4a4878dc19562163c596b"},{"pmid_author_id":38116971,"last_name":"Sanaei","first_name":"Sara","initials":"S","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"3335f834045c1f1738d62f737a3f0873"},{"pmid_author_id":38116972,"last_name":"Eskandari","first_name":"Ebrahim","initials":"E","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"657f3bd70662e691a67ecbbc8d351cc4"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"}]}]},{"RECORD":{"pmid_entry_id":27687212,"pmid":29048575,"publication_date":"1969-12-31 00:00:00","title":"MicroRNA-binding site polymorphisms in genes involved in colorectal cancer etiopathogenesis and their impact on disease prognosis.","doi":"10.1093\/mutage\/gex026","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":4452,"month_1910":-719,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":4452,"journal_name":"Mutagenesis","journal_abbr":"Mutagenesis","issn_print":"0267-8357","issn_online":"1464-3804","iso_abbr":"Mutagenesis","nlmid":"8707812"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":106083580,"pmid_entry_id":27687212,"pmid_author_id":23902022,"position":14},{"pmid_author_map_id":106083579,"pmid_entry_id":27687212,"pmid_author_id":8953691,"position":13},{"pmid_author_map_id":106083578,"pmid_entry_id":27687212,"pmid_author_id":39803002,"position":12},{"pmid_author_map_id":106083577,"pmid_entry_id":27687212,"pmid_author_id":39803001,"position":11},{"pmid_author_map_id":106083576,"pmid_entry_id":27687212,"pmid_author_id":39803000,"position":10},{"pmid_author_map_id":106083575,"pmid_entry_id":27687212,"pmid_author_id":39802999,"position":9},{"pmid_author_map_id":106083574,"pmid_entry_id":27687212,"pmid_author_id":39802998,"position":8},{"pmid_author_map_id":106083573,"pmid_entry_id":27687212,"pmid_author_id":29392295,"position":7},{"pmid_author_map_id":106083572,"pmid_entry_id":27687212,"pmid_author_id":31343320,"position":6},{"pmid_author_map_id":106083571,"pmid_entry_id":27687212,"pmid_author_id":39611616,"position":5},{"pmid_author_map_id":106083570,"pmid_entry_id":27687212,"pmid_author_id":39802997,"position":4},{"pmid_author_map_id":106083569,"pmid_entry_id":27687212,"pmid_author_id":39802996,"position":3},{"pmid_author_map_id":106083568,"pmid_entry_id":27687212,"pmid_author_id":8798076,"position":2},{"pmid_author_map_id":106083567,"pmid_entry_id":27687212,"pmid_author_id":26533255,"position":1}],"pmid_abstract":[{"pmid_abstract_id":29563967,"pmid_entry_id":27687212,"abstract_type":"Text","abstract_text":"According to the Vogelstein's model of colorectal carcinogenesis, genetic variations in highly penetrant genes may be involved in the colorectal cancer (CRC) pathogenesis. Similarly, aberrant function and\/or altered expression of microRNAs (miRNAs) often occur in CRC. In this context, polymorphisms in miRNA-binding sites (miRSNPs) may affect miRNA\/target gene interaction, resulting in differential mRNA\/protein expression and increased susceptibility to common diseases. To explore this phenomenon, we have mined the 3' untranslated regions (3'UTRs) of genes known to be frequently mutated in CRC to search for miRSNPs and tested their association with CRC risk and clinical outcome. Eight miRSNPs (rs1804191, rs397768, rs41116 in APC; rs1137918, s227091, rs4585 in ATM; rs712, rs1137282, rs61764370 in KRAS; rs8674 in PARP1 and rs16950113 in SMAD7) were tested for their association with CRC risk in a case-control study (1111 cases and 1469 healthy controls). The role of these miRSNPs was also investigated in relation to clinical outcome on a subset of patients with complete follow-up. rs8679 within PARP1 was associated with CRC risk and patients' survival. In the dominant model, carriers of at least one C allele were at a decreased risk of cancer (P = 0.05). The CC genotype in rs8679 was also associated with an increased risk of recurrence\/progression in patients that received 5-FU-based chemotherapy (log-rank test P = 0.03). Carriers of the homozygous variant genotype TT for rs712 in KRAS gene were associated with a decreased risk of rectal cancer (odds ratio (OR) = 0.65, 95% confidence intervals (CI) 0.43-1.00, P = 0.05) while individuals with colon cancer carrying the heterozygous GT genotype showed a longer overall survival (OS) (P = 0.04). We provide the first evidence that variations in potential miRNA-binding target sites in the 3' UTR of PARP1 gene may modulate CRC risk and prognosis after therapy. Further studies are needed to replicate our finding and assess miRSNPs as predictive biomarkers in independent populations."}]},{"pmid_author":[{"pmid_author_id":8798076,"last_name":"Naccarati","first_name":"Alessio","initials":"A","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"f230aa25ae234902697adcb69141a25a"},{"pmid_author_id":8953691,"last_name":"Vodicka","first_name":"Pavel","initials":"P","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"3dd25dd518835058bf8b5ec8d0253ac8"},{"pmid_author_id":23902022,"last_name":"Vymetalkova","first_name":"Veronika","initials":"V","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"18d8455ad6ad4682b66bf6a068770ca7"},{"pmid_author_id":26533255,"last_name":"Schneiderova","first_name":"Michaela","initials":"M","is_valid_author":"Y","pmid_instit_id":10799564,"orcid_id":null,"md5_hash":"4d79da3837cdfde85bbf0d92bc7ddfe0"},{"pmid_author_id":29392295,"last_name":"Opattova","first_name":"Alena","initials":"A","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"7048ba9132f89a1262568ad42864dd3f"},{"pmid_author_id":31343320,"last_name":"Jiraskova","first_name":"Katerina","initials":"K","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8de112468d89d8f843bb6383d99d4a03"},{"pmid_author_id":39611616,"last_name":"Gaetano","first_name":"Cornelia Di","initials":"CD","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"b951bd1676414d68c5941547efd44006"},{"pmid_author_id":39802996,"last_name":"Pardini","first_name":"Barbara","initials":"B","is_valid_author":"Y","pmid_instit_id":16810302,"orcid_id":null,"md5_hash":"582d5fbfbb53f01809ea8bc077d82e61"},{"pmid_author_id":39802997,"last_name":"Rosa","first_name":"Fabio","initials":"F","is_valid_author":"Y","pmid_instit_id":16810302,"orcid_id":null,"md5_hash":"af8a32edf2483d18e3a94536494f0a14"},{"pmid_author_id":39802998,"last_name":"Levy","first_name":"Miroslav","initials":"M","is_valid_author":"Y","pmid_instit_id":16810303,"orcid_id":null,"md5_hash":"a502ef645d88b059846fd006eed2190e"},{"pmid_author_id":39802999,"last_name":"Veskrna","first_name":"Karel","initials":"K","is_valid_author":"Y","pmid_instit_id":16810303,"orcid_id":null,"md5_hash":"113927422d0ce230507e1c1075f5763e"},{"pmid_author_id":39803000,"last_name":"Veskrnova","first_name":"Veronika","initials":"V","is_valid_author":"Y","pmid_instit_id":16810304,"orcid_id":null,"md5_hash":"ab6682af058656003a373d10741202ed"},{"pmid_author_id":39803001,"last_name":"Buchler","first_name":"Tomas","initials":"T","is_valid_author":"Y","pmid_instit_id":16810304,"orcid_id":null,"md5_hash":"32de9d2785975a80a3bf4e3c2ea16ce6"},{"pmid_author_id":39803002,"last_name":"Landi","first_name":"Stefano","initials":"S","is_valid_author":"Y","pmid_instit_id":16200706,"orcid_id":null,"md5_hash":"c5f05e42b8e39abafe05f386909c03a2"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":10799564,"instit_name":"Department of Surgery, General University Hospital in Prague, Prague 12800, Czech Republic.","instit_hash":"c2e343eb6851bd6b4c7bf4af4f67858a"},{"pmid_instit_id":16200706,"instit_name":"Department of Biology, University of Pisa, Via Derna 1, 56126 Pisa, Italy.","instit_hash":"effd6ae82cac5b945a69399bdb2132b9"},{"pmid_instit_id":16810302,"instit_name":"Molecular and Genetic Epidemiology; Genomic Variation in Human Populations and Complex Diseases, IIGM Italian Institute for Genomic Medicine, Via Nizza 52, 10126 Turin, Italy.","instit_hash":"fcebcc7441b4aa73dd6bf2f078d31d72"},{"pmid_instit_id":16810303,"instit_name":"Department of Surgery, First Faculty of Medicine, Charles University and Thomayer Hospital, Videnska 800, 140 59 Prague, Czech Republic.","instit_hash":"b4cd2c88e43382ce44c110dc7f3cebc3"},{"pmid_instit_id":16810304,"instit_name":"Department of Oncology, First Faculty of Medicine, Charles University and Thomayer Hospital, Videnska 800, 140 59 Prague, Czech Republic.","instit_hash":"09d13d9d17a8a24c2e6881635751a001"}]}]},{"RECORD":{"pmid_entry_id":28575645,"pmid":29963045,"publication_date":"2018-01-01 00:00:00","title":"MicroRNA-Related Polymorphisms in Infectious Diseases-Tiny Changes With a Huge Impact on Viral Infections and Potential Clinical Applications.","doi":"10.3389\/fimmu.2018.01316","volume":"9","issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":26594,"month_1910":-1296,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":26594,"journal_name":"Frontiers in immunology","journal_abbr":"Front Immunol","issn_print":"","issn_online":"1664-3224","iso_abbr":"Front Immunol","nlmid":"101560960"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":110952040,"pmid_entry_id":28575645,"pmid_author_id":43321323,"position":4},{"pmid_author_map_id":110952039,"pmid_entry_id":28575645,"pmid_author_id":16108769,"position":3},{"pmid_author_map_id":110952038,"pmid_entry_id":28575645,"pmid_author_id":43321322,"position":2},{"pmid_author_map_id":110952037,"pmid_entry_id":28575645,"pmid_author_id":43321321,"position":1}],"pmid_abstract":[{"pmid_abstract_id":31048972,"pmid_entry_id":28575645,"abstract_type":"Text","abstract_text":"MicroRNAs (miRNAs) are single-stranded sequences of non-coding RNA with approximately 22 nucleotides that act posttranscriptionally on gene expression. miRNAs are important gene regulators in physiological contexts, but they also impact the pathogenesis of various diseases. The role of miRNAs in viral infections has been explored by different authors in both population-based as well as in functional studies. However, the effect of miRNA polymorphisms on the susceptibility to viral infections and on the clinical course of these diseases is still an emerging topic. Thus, this review will compile and organize the findings described in studies that evaluated the effects of genetic variations on miRNA genes and on their binding sites, in the context of human viral diseases. In addition to discussing the basic aspects of miRNAs biology, we will cover the studies that investigated miRNA polymorphisms in infections caused by hepatitis B virus, hepatitis C virus, human immunodeficiency virus, Epstein-Barr virus, and human papillomavirus. Finally, emerging topics concerning the importance of miRNA genetic variants will be presented, focusing on the context of viral infectious diseases."}]},{"pmid_author":[{"pmid_author_id":16108769,"last_name":"Guimar\u00e3es","first_name":"Rafael Lima","initials":"RL","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"45e6fcf42edcd4b3665792eea0b9f4fb"},{"pmid_author_id":43321321,"last_name":"Ellwanger","first_name":"Joel Henrique","initials":"JH","is_valid_author":"Y","pmid_instit_id":18301919,"orcid_id":null,"md5_hash":"104e0fc3533cb1a12723df7fc937f887"},{"pmid_author_id":43321322,"last_name":"Zambra","first_name":"Francis Maria B\u00e1o","initials":"FMB","is_valid_author":"Y","pmid_instit_id":18301919,"orcid_id":null,"md5_hash":"f7bff750305221d6fe42ab1f19231771"},{"pmid_author_id":43321323,"last_name":"Chies","first_name":"Jos\u00e9 Artur Bogo","initials":"JAB","is_valid_author":"Y","pmid_instit_id":18301919,"orcid_id":null,"md5_hash":"22b82844941e8330c1ecaa8ef3ed02dc"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":18301919,"instit_name":"Laborat\u00f3rio de Imunobiologia e Imunogen\u00e9tica, Programa de P\u00f3s-Gradua\u00e7\u00e3o em Gen\u00e9tica e Biologia Molecular, Departamento de Gen\u00e9tica, Universidade Federal do Rio Grande do Sul (UFRGS), Porto Alegre, Brazil.","instit_hash":"e8f7fce4173fc1108d0b3113e2d4c977"}]}]},{"RECORD":{"pmid_entry_id":28941614,"pmid":30337837,"publication_date":"2018-01-01 00:00:00","title":"Single nucleotide polymorphisms and sporadic colorectal cancer susceptibility: a field synopsis and meta-analysis.","doi":"10.1186\/s12935-018-0656-2","volume":"18","issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":17431,"month_1910":-1296,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":17431,"journal_name":"Cancer cell international","journal_abbr":"Cancer Cell Int","issn_print":"","issn_online":"1475-2867","iso_abbr":"Cancer Cell Int","nlmid":"101139795"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":113052889,"pmid_entry_id":28941614,"pmid_author_id":7947162,"position":3},{"pmid_author_map_id":113052888,"pmid_entry_id":28941614,"pmid_author_id":9236933,"position":2},{"pmid_author_map_id":113052887,"pmid_entry_id":28941614,"pmid_author_id":7635412,"position":1}],"pmid_abstract":[{"pmid_abstract_id":31688384,"pmid_entry_id":28941614,"abstract_type":"Background","abstract_text":"Although mounting non-hereditary colorectal cancer (NHCRC) associated single nucleotide polymorphisms (SNPs) have been observed, no field synopsis and meta-analysis has been conducted through systematically assessing cumulative evidence, during the past 5\u00a0years."},{"pmid_abstract_id":31688385,"pmid_entry_id":28941614,"abstract_type":"Methods","abstract_text":"We retrieved the database via the PubMed, Web of Science and Embase gateways to identify publications concerning the associations between SNPs and risk of NHCRC, up to May 1st, 2017. To assess the finding credibility, cumulative evidence was graded based on the Venice criteria. Meta-analysis was also performed for three subgroups including ethnicity (Asian vs Caucasian), primary cancer site (colon vs rectum) and TNM stage (I II vs III IV). Then, we arranged those high quality SNPs into different regions according to their locations on genes to evaluate their functional roles on CRC development."},{"pmid_abstract_id":31688386,"pmid_entry_id":28941614,"abstract_type":"Results","abstract_text":"5114 publications were collected and 1001 of them met our inclusion criteria, which totally included 1788 SNPs in 793 genes or distinct chromosomal loci. Totally, we performed 359 primary and subgroup meta-analyses for 160 SNPs in 96 distinct genes. By utilizing the Venice criteria, we identified 15 high quality SNPs with 25 high credibility significant associations. Furthermore, we artificially divided the high quality SNPs into different groups, based on their SNP loci (exon region, intron region, promoter region, downstream region, non-coding region and intergenic region)."},{"pmid_abstract_id":31688387,"pmid_entry_id":28941614,"abstract_type":"Conclusion","abstract_text":"We have identified 15 high quality SNPs which may act as promising genetic biomarkers for clinical NHCRC susceptibility screening and explored their functional roles on the NHCRC development based on their locations on genes."},{"pmid_abstract_id":40685491,"pmid_entry_id":28941614,"abstract_type":"BACKGROUND","abstract_text":"Although mounting non-hereditary colorectal cancer (NHCRC) associated single nucleotide polymorphisms (SNPs) have been observed, no field synopsis and meta-analysis has been conducted through systematically assessing cumulative evidence, during the past 5\u00a0years."},{"pmid_abstract_id":40685492,"pmid_entry_id":28941614,"abstract_type":"METHODS","abstract_text":"We retrieved the database via the PubMed, Web of Science and Embase gateways to identify publications concerning the associations between SNPs and risk of NHCRC, up to May 1st, 2017. To assess the finding credibility, cumulative evidence was graded based on the Venice criteria. Meta-analysis was also performed for three subgroups including ethnicity (Asian vs Caucasian), primary cancer site (colon vs rectum) and TNM stage (I II vs III IV). Then, we arranged those high quality SNPs into different regions according to their locations on genes to evaluate their functional roles on CRC development."},{"pmid_abstract_id":40685493,"pmid_entry_id":28941614,"abstract_type":"RESULTS","abstract_text":"5114 publications were collected and 1001 of them met our inclusion criteria, which totally included 1788 SNPs in 793 genes or distinct chromosomal loci. Totally, we performed 359 primary and subgroup meta-analyses for 160 SNPs in 96 distinct genes. By utilizing the Venice criteria, we identified 15 high quality SNPs with 25 high credibility significant associations. Furthermore, we artificially divided the high quality SNPs into different groups, based on their SNP loci (exon region, intron region, promoter region, downstream region, non-coding region and intergenic region)."},{"pmid_abstract_id":40685494,"pmid_entry_id":28941614,"abstract_type":"CONCLUSION","abstract_text":"We have identified 15 high quality SNPs which may act as promising genetic biomarkers for clinical NHCRC susceptibility screening and explored their functional roles on the NHCRC development based on their locations on genes."}]},{"pmid_author":[{"pmid_author_id":7635412,"last_name":"Wen","first_name":"Jing","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"0720fbf856f8cd9323231f55dd4f900f"},{"pmid_author_id":7947162,"last_name":"Yuan","first_name":"Yuan","initials":"Y","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"dff7ca5a7f9a04927bb8969832d87ae9"},{"pmid_author_id":9236933,"last_name":"Xu","first_name":"Qian","initials":"Q","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"eb3da06e80e7dd4bc89327d080f72c1c"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"}]}]},{"RECORD":{"pmid_entry_id":29739025,"pmid":31152438,"publication_date":"1969-12-31 00:00:00","title":"A single nuclear polymorphism in let-7g binding site affects the doubling time of thyroid nodule by regulating KRAS-induced cell proliferation.","doi":"10.1002\/jcp.28912","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":122,"month_1910":-719,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":122,"journal_name":"Journal of cellular physiology","journal_abbr":"J Cell Physiol","issn_print":"0021-9541","issn_online":"1097-4652","iso_abbr":"J Cell Physiol","nlmid":"0050222"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":117738627,"pmid_entry_id":29739025,"pmid_author_id":48166985,"position":6},{"pmid_author_map_id":117738626,"pmid_entry_id":29739025,"pmid_author_id":48166984,"position":5},{"pmid_author_map_id":117738625,"pmid_entry_id":29739025,"pmid_author_id":48166983,"position":4},{"pmid_author_map_id":117738624,"pmid_entry_id":29739025,"pmid_author_id":48166982,"position":3},{"pmid_author_map_id":117738623,"pmid_entry_id":29739025,"pmid_author_id":46758535,"position":2},{"pmid_author_map_id":117738622,"pmid_entry_id":29739025,"pmid_author_id":48166981,"position":1}],"pmid_abstract":[{"pmid_abstract_id":33061462,"pmid_entry_id":29739025,"abstract_type":"Text","abstract_text":"As an indicator for the malignancy of thyroid nodules (TN), the doubling time of TN was studied in this study to evaluate the effect of rs712 polymorphism on the progression of TN. In addition, we aimed to study the potential molecular mechanisms underlying the pathological effect of rs712 polymorphism upon TN. A Taqman method was used to genotype the patients according to their rs712 polymorphism. Real-time polymerase chain reaction, western blot, Terminal deoxynucleotidyl transferase dUTP nick end labeling assay and 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium (MTT) assay was conducted to study the correlation between KRAS expression and the pathological effect of rs712 polymorphism. In-silicon analysis and luciferase assay were utilized to establish the regulatory relationship between let-7g and KRAS. KRAS messenger RNA (mRNA)\/protein levels in the GG group were upregulated with a decreased apoptosis index. KRAS mRNA was validated to be a virtual target of let-7g. In addition, the mRNA\/protein level of KRAS as well as cell proliferation index was decreased in primary thyroid cancer cells genotyped as TT\/TG and transfected with KRAS small interfering RNA (siRNA)\/let-7g precursors. The cell apoptosis index was evidently elevated in the KRAS siRNA\/let-7g precursors group compared with that in the scramble controls. Moreover, KRAS mRNA\/protein only showed slight reduction when GG-genotyped primary thyroid cancer cells were transfected by let-7g precursors. Additionally, let-7g precursors exhibited no significant effect on cell proliferation index or cell apoptosis in GG cells. Rs712 polymorphism T>G in the 3'-untranslated region of KRAS interrupts the interactions between let-7g and KRAS mRNA, leading to a higher cell proliferation index and reduced doubling time of TN."}]},{"pmid_author":[{"pmid_author_id":46758535,"last_name":"Zhang","first_name":"Wanli","initials":"W","is_valid_author":"Y","pmid_instit_id":6516076,"orcid_id":null,"md5_hash":"3f705ca0cdfbe5cec597b9756652cb64"},{"pmid_author_id":48166981,"last_name":"Liu","first_name":"Ailin","initials":"A","is_valid_author":"Y","pmid_instit_id":20380236,"orcid_id":null,"md5_hash":"9a83bd79b9c843d7cc46f24cbc208fa4"},{"pmid_author_id":48166982,"last_name":"Zhao","first_name":"Tao","initials":"T","is_valid_author":"Y","pmid_instit_id":20380236,"orcid_id":null,"md5_hash":"b937160e7b505ce2ad5e7660d30af4fb"},{"pmid_author_id":48166983,"last_name":"Xiao","first_name":"Ming","initials":"M","is_valid_author":"Y","pmid_instit_id":20380237,"orcid_id":null,"md5_hash":"89678dabc34a96513042645b72b23180"},{"pmid_author_id":48166984,"last_name":"Mei","first_name":"Qijian","initials":"Q","is_valid_author":"Y","pmid_instit_id":20380237,"orcid_id":null,"md5_hash":"807446fe22bb24a0da20606d690f275b"},{"pmid_author_id":48166985,"last_name":"Zhu","first_name":"Hui","initials":"H","is_valid_author":"Y","pmid_instit_id":20380237,"orcid_id":"0000-0002-5078-9404","md5_hash":"aa254447051f70fef8cc4ed83640f5ba"}]},{"pmid_instit":[{"pmid_instit_id":6516076,"instit_name":"Cancer Center, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China.","instit_hash":"107e1369554006c0af88a8acedabde8a"},{"pmid_instit_id":20380236,"instit_name":"Department of Endocrinology, Xiaogan Hospital Affiliated to Wuhan University of Science and Technology, Xiaogan, China.","instit_hash":"d18c6717930730da0c9d46fc1261d4cf"},{"pmid_instit_id":20380237,"instit_name":"Department of Geratology, General Hospital of the Yangtze River Shipping, Wuhan, China.","instit_hash":"b8e9d3f6d0ee9f66a98fd193cf644b83"}]}]},{"RECORD":{"pmid_entry_id":29743193,"pmid":31156795,"publication_date":"2019-03-01 00:00:00","title":"Association of rs712 polymorphism in a let-7 microRNA-binding site of KRAS<\/i> gene with colorectal cancer in a Mexican population.","doi":"10.22038\/ijbms.2019.26564.6507","volume":"22","issue":"3","pages":null,"pmid_status":"ppublish","pmid_journal_id":27921,"month_1910":-1310,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":27921,"journal_name":"Iranian journal of basic medical sciences","journal_abbr":"Iran J Basic Med Sci","issn_print":"2008-3866","issn_online":"2008-3874","iso_abbr":"Iran J Basic Med Sci","nlmid":"101517966"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":117763250,"pmid_entry_id":29743193,"pmid_author_id":48185826,"position":7},{"pmid_author_map_id":117763249,"pmid_entry_id":29743193,"pmid_author_id":48185825,"position":6},{"pmid_author_map_id":117763248,"pmid_entry_id":29743193,"pmid_author_id":48185824,"position":5},{"pmid_author_map_id":117763247,"pmid_entry_id":29743193,"pmid_author_id":48185823,"position":4},{"pmid_author_map_id":117763246,"pmid_entry_id":29743193,"pmid_author_id":48185822,"position":3},{"pmid_author_map_id":117763245,"pmid_entry_id":29743193,"pmid_author_id":48185821,"position":2},{"pmid_author_map_id":117763244,"pmid_entry_id":29743193,"pmid_author_id":48185820,"position":1}],"pmid_abstract":[{"pmid_abstract_id":33068801,"pmid_entry_id":29743193,"abstract_type":"Objectives","abstract_text":"The rs712 polymorphism in a let-7 microRNA-binding site at gene has been associated with cancer. To examine its association with rs712 polymorphism, we analyzed Mexican individuals with colorectal cancer (CRC) and healthy subjects."},{"pmid_abstract_id":33068802,"pmid_entry_id":29743193,"abstract_type":"Materials and Methods","abstract_text":"Genotyping of the rs712 polymorphism was performed by polymerase chain reaction in 281 controls and 336 CRC patients."},{"pmid_abstract_id":33068803,"pmid_entry_id":29743193,"abstract_type":"Results","abstract_text":"The observed frequencies of rs712 polymorphism indicated an associated protective factor for CRC (=0.032). An association between genotype and the disease was evident in: colon localization (allele odds ratio (OR) 3.82, 95% confidence Intervals (CI) 2.77-5.28, =0.0001), node metastasis (genotype OR 2.49, 95% CI 1.45-4.28, =0.0009), poor differentiation (genotype GT, OR 2.35, 95% CI 1.35-4.1, =0.0033), and poor chemotherapy response (genotype GT, OR 2.6, 95% CI 1.7-4.24, =0.0001)."},{"pmid_abstract_id":33068804,"pmid_entry_id":29743193,"abstract_type":"Conclusion","abstract_text":"Comparison of the data from patients with control group showed that polymorphism of rs712 in gene was protective factor, which was associated with susceptibility for CRC. However, the genotypes and of rs712 polymorphism in could contribute significantly to colon localization, node metastasis, poor differentiation and poor chemotherapy response in CRC patients in this sample population."},{"pmid_abstract_id":40981361,"pmid_entry_id":29743193,"abstract_type":"OBJECTIVES","abstract_text":"The rs712 polymorphism in a let-7 microRNA-binding site at gene has been associated with cancer. To examine its association with rs712 polymorphism, we analyzed Mexican individuals with colorectal cancer (CRC) and healthy subjects."},{"pmid_abstract_id":40981362,"pmid_entry_id":29743193,"abstract_type":"MATERIALS AND METHODS","abstract_text":"Genotyping of the rs712 polymorphism was performed by polymerase chain reaction in 281 controls and 336 CRC patients."},{"pmid_abstract_id":40981363,"pmid_entry_id":29743193,"abstract_type":"RESULTS","abstract_text":"The observed frequencies of rs712 polymorphism indicated an associated protective factor for CRC (=0.032). An association between genotype and the disease was evident in: colon localization (allele odds ratio (OR) 3.82, 95% confidence Intervals (CI) 2.77-5.28, =0.0001), node metastasis (genotype OR 2.49, 95% CI 1.45-4.28, =0.0009), poor differentiation (genotype GT, OR 2.35, 95% CI 1.35-4.1, =0.0033), and poor chemotherapy response (genotype GT, OR 2.6, 95% CI 1.7-4.24, =0.0001)."},{"pmid_abstract_id":40981364,"pmid_entry_id":29743193,"abstract_type":"CONCLUSION","abstract_text":"Comparison of the data from patients with control group showed that polymorphism of rs712 in gene was protective factor, which was associated with susceptibility for CRC. However, the genotypes and of rs712 polymorphism in could contribute significantly to colon localization, node metastasis, poor differentiation and poor chemotherapy response in CRC patients in this sample population."}]},{"pmid_author":[{"pmid_author_id":48185820,"last_name":"Gallegos-Arreola","first_name":"Martha Patricia","initials":"MP","is_valid_author":"Y","pmid_instit_id":1323502,"orcid_id":null,"md5_hash":"7ab101f0d64f4cdd91f908ea4599f6a5"},{"pmid_author_id":48185821,"last_name":"Z\u00fa\u00f1iga-Gonz\u00e1lez","first_name":"Guillermo Mois\u00e9s","initials":"GM","is_valid_author":"Y","pmid_instit_id":20388613,"orcid_id":null,"md5_hash":"e5fdee65dcf20591b77e6822ce5dff00"},{"pmid_author_id":48185822,"last_name":"G\u00f3mez-Mariscal","first_name":"Karen","initials":"K","is_valid_author":"Y","pmid_instit_id":20388613,"orcid_id":null,"md5_hash":"b8eb0e8ad784db3e2e2f359e1294f8ab"},{"pmid_author_id":48185823,"last_name":"Rosales-Reynoso","first_name":"M\u00f3nica Alejandra","initials":"MA","is_valid_author":"Y","pmid_instit_id":20388613,"orcid_id":null,"md5_hash":"a5daf6283878aa06190ffab399599e33"},{"pmid_author_id":48185824,"last_name":"Luis","first_name":"Luis","initials":"L","is_valid_author":"Y","pmid_instit_id":1323502,"orcid_id":null,"md5_hash":"b9658ef7bc2b92584e0e064d8083950f"},{"pmid_author_id":48185825,"last_name":"Puebla-P\u00e9rez","first_name":"Ana Mar\u00eda","initials":"AM","is_valid_author":"Y","pmid_instit_id":20388614,"orcid_id":null,"md5_hash":"47084080e103edd08290524d5dc459f0"},{"pmid_author_id":48185826,"last_name":"Pineda-Razo","first_name":"Tomas","initials":"T","is_valid_author":"Y","pmid_instit_id":20388615,"orcid_id":null,"md5_hash":"2f71ce26f126cb4cdc8f326bbc411a54"}]},{"pmid_instit":[{"pmid_instit_id":1323502,"instit_name":"Divisi\u00f3n de Gen\u00e9tica, Centro de Investigaci\u00f3n Biom\u00e9dica de Occidente, Instituto Mexicano del Seguro Social, Guadalajara, Jalisco, M\u00e9xico.","instit_hash":"0ec0303d8f43afc20b8f1d9d1f9bd93c"},{"pmid_instit_id":20388613,"instit_name":"Medicina Molecular, Centro de Investigaci\u00f3n Biom\u00e9dica de Occidente, Instituto Mexicano del Seguro Social, Guadalajara, Jalisco, Mexico.","instit_hash":"17563e7c14793fe2d5ab5997f1c8cb0c"},{"pmid_instit_id":20388614,"instit_name":"Laboratorio de Inmunofarmacolog\u00eda, Centro Universitario de Ciencias Exactas e Ingenier\u00edas, Universidad de Guadalajara, Guadalajara, Jalisco, Mexico.","instit_hash":"579860f1b783adfb8a587c1e1bc2d472"},{"pmid_instit_id":20388615,"instit_name":"Servicio de Oncolog\u00eda, Unidad M\u00e9dica de Alta Especialidad, Hospital de Especialidades, Centro M\u00e9dico Nacional de Occidente, Instituto Mexicano del Seguro Social, Guadalajara, Jalisco, Mexico.","instit_hash":"e7690da4d3446b67bf864053f5fe3e47"}]}]},{"RECORD":{"pmid_entry_id":29851898,"pmid":31269493,"publication_date":"2020-01-01 00:00:00","title":"Single Nucleotide Polymorphisms in MicroRNA-Binding Site of Epidermal Growth Factor Receptor Signaling Pathway and Susceptibility to Esophageal Squamous Cell Carcinoma.","doi":"10.1159\/000501447","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":4738,"month_1910":-1320,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":4738,"journal_name":"Digestive diseases (Basel, Switzerland)","journal_abbr":"Dig Dis","issn_print":"0257-2753","issn_online":"1421-9875","iso_abbr":"Dig Dis","nlmid":"8701186"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":118449167,"pmid_entry_id":29851898,"pmid_author_id":15584010,"position":8},{"pmid_author_map_id":118449166,"pmid_entry_id":29851898,"pmid_author_id":7943372,"position":7},{"pmid_author_map_id":118449165,"pmid_entry_id":29851898,"pmid_author_id":48654668,"position":6},{"pmid_author_map_id":118449164,"pmid_entry_id":29851898,"pmid_author_id":48654667,"position":5},{"pmid_author_map_id":118449163,"pmid_entry_id":29851898,"pmid_author_id":48654666,"position":4},{"pmid_author_map_id":118449162,"pmid_entry_id":29851898,"pmid_author_id":48654665,"position":3},{"pmid_author_map_id":118449161,"pmid_entry_id":29851898,"pmid_author_id":48654664,"position":2},{"pmid_author_map_id":118449160,"pmid_entry_id":29851898,"pmid_author_id":48654663,"position":1}],"pmid_abstract":[{"pmid_abstract_id":33259114,"pmid_entry_id":29851898,"abstract_type":"BACKGROUND","abstract_text":"Epidermal growth factor receptor (EGFR) signaling pathway plays a fundamental role in regulating cell proliferation, differentiation, apoptosis, migration, and so on, which are associated with tumor development, including the esophageal squamous cell carcinoma (ESCC). The single nucleotide polymorphisms (SNPs) of microRNA-binding sites with target genes in the EGFR pathway could lead to alteration in the combination of microRNA with target genes and contribute to the susceptibility of cancer."},{"pmid_abstract_id":33259115,"pmid_entry_id":29851898,"abstract_type":"METHODS","abstract_text":"A case-control study including 494 ESCC patients and 494 controls was carried out to investigate the genetic susceptibility of 4 microRNA-binding site SNPs (rs712 in the binding site of KRAS to let-7, rs8904 in the binding site of NFBIA to mir-507, rs3738894 in the binding site of protein kinase C epsilon to mir-218, rs701848 in the binding site of phosphatase and tensin to mir-1304) as well as the interactions of gene-environment in the development of ESCC."},{"pmid_abstract_id":33259116,"pmid_entry_id":29851898,"abstract_type":"RESULTS","abstract_text":"The results showed that compared with CC genotype, the individuals with TT and TT + CT genotypes of rs701848 were significantly associated with increased ESCC risk (OR adjusted 1.56, 95% CI 1.07-2.27 and 1.41, 1.01-1.97). The gene-Environment interaction between rs3738894 and smoking history was associated with the risk of esophageal cancer."},{"pmid_abstract_id":33259117,"pmid_entry_id":29851898,"abstract_type":"CONCLUSIONS","abstract_text":"Results of these analyses underline the support of the notion that SNPs in microRNA-binding site of EGFR signaling pathway play certain important roles in the susceptibility to ESCC."}]},{"pmid_author":[{"pmid_author_id":7943372,"last_name":"Zhang","first_name":"Jianying","initials":"J","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"639a53ca888bad987044029bb7a92446"},{"pmid_author_id":15584010,"last_name":"Dai","first_name":"Liping","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"931b68e4380ebb0122fd4f15ae1c5ec6"},{"pmid_author_id":48654663,"last_name":"Qu","first_name":"Yanhong","initials":"Y","is_valid_author":"Y","pmid_instit_id":6467165,"orcid_id":null,"md5_hash":"2e5b649e2eb8ee381a82a077a7ce3cea"},{"pmid_author_id":48654664,"last_name":"Zhang","first_name":"Yougai","initials":"Y","is_valid_author":"Y","pmid_instit_id":20586891,"orcid_id":null,"md5_hash":"c6661da6798eba45e032ffdb32d5f972"},{"pmid_author_id":48654665,"last_name":"Wang","first_name":"Kaijuan","initials":"K","is_valid_author":"Y","pmid_instit_id":20586892,"orcid_id":null,"md5_hash":"5483b7364316aa489f991efd72aead0a"},{"pmid_author_id":48654666,"last_name":"Song","first_name":"Chunhua","initials":"C","is_valid_author":"Y","pmid_instit_id":20586892,"orcid_id":null,"md5_hash":"895fbc8bdfdce8373c647c0d577740bc"},{"pmid_author_id":48654667,"last_name":"Wang","first_name":"Peng","initials":"P","is_valid_author":"Y","pmid_instit_id":20586892,"orcid_id":null,"md5_hash":"d631a8833efe9dd444f615ee133be732"},{"pmid_author_id":48654668,"last_name":"Ye","first_name":"Hua","initials":"H","is_valid_author":"Y","pmid_instit_id":20586892,"orcid_id":null,"md5_hash":"381065fe3540b464ef489d5f536b5e5c"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":6467165,"instit_name":"The Third Affiliated Hospital, Zhengzhou University, Zhengzhou, China.","instit_hash":"941cd43142433d2e4a1d5f3066bdfc22"},{"pmid_instit_id":20586891,"instit_name":"The Affiliated Tumor Hospital, Zhengzhou University, Zhengzhou, China.","instit_hash":"7e4e62bf42446e6d22a7ca720b72538a"},{"pmid_instit_id":20586892,"instit_name":"Henan Key Laboratory of Tumor Epidemiology, Zhenghzou University, Zhengzhou, China.","instit_hash":"d426674eeda6351cfd04da78d54efeca"}]}]},{"RECORD":{"pmid_entry_id":30212889,"pmid":31637880,"publication_date":"1969-12-31 00:00:00","title":"MicroRNA-binding site polymorphisms and risk of colorectal cancer: A systematic review and meta-analysis.","doi":"10.1002\/cam4.2600","volume":null,"issue":null,"pages":null,"pmid_status":"ppublish","pmid_journal_id":28080,"month_1910":-719,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":28080,"journal_name":"Cancer medicine","journal_abbr":"Cancer Med","issn_print":"","issn_online":"2045-7634","iso_abbr":"Cancer Med","nlmid":"101595310"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":171030459,"pmid_entry_id":30212889,"pmid_author_id":84547867,"position":1},{"pmid_author_map_id":120616001,"pmid_entry_id":30212889,"pmid_author_id":50220965,"position":8},{"pmid_author_map_id":120616000,"pmid_entry_id":30212889,"pmid_author_id":50220964,"position":7},{"pmid_author_map_id":120615999,"pmid_entry_id":30212889,"pmid_author_id":34083804,"position":6},{"pmid_author_map_id":120615998,"pmid_entry_id":30212889,"pmid_author_id":24626483,"position":5},{"pmid_author_map_id":120615997,"pmid_entry_id":30212889,"pmid_author_id":45615941,"position":4},{"pmid_author_map_id":120615996,"pmid_entry_id":30212889,"pmid_author_id":24626884,"position":3},{"pmid_author_map_id":120615995,"pmid_entry_id":30212889,"pmid_author_id":24626885,"position":2},{"pmid_author_map_id":120615994,"pmid_entry_id":30212889,"pmid_author_id":20043398,"position":1}],"pmid_abstract":[{"pmid_abstract_id":33898077,"pmid_entry_id":30212889,"abstract_type":"Text","abstract_text":"Genetic variations in miRNAs binding site might participate in cancer risk. This study aimed to systematically review the association between miRNA-binding site polymorphisms and colorectal cancer (CRC). Electronic literature search was carried out on PubMed, Web of Science (WOS), Scopus, and Embase. All types of observational studies till 30 November 2018 were included. Overall 85 studies (21 SNPs) from two systematic searches were included analysis. The results showed that in the Middle East population, the minor allele of rs731236 was associated with decreased risk of CRC (heterozygote model: 0.76 [0.61-0.95]). The minor allele of rs3025039 was related to increased risk of CRC in East Asian population (allelic model: 1.25 [1.01-1.54]). Results for rs3212986 were significant in overall and subgroup analysis (P\u00a0<\u00a0.05). For rs1801157 in subgroup analysis the association was significant in Asian populations (including allelic model: 2.28 [1.11-4.69]). For rs712, subgroup analysis revealed a significant (allelic model: 1.41 [1.23-1.61]) and borderline (allelic model: 0.92 [0.84-1.00]) association in Chinese and Czech populations, respectively. The minor allele of rs17281995 increased risk of CRC in different genetic models (P\u00a0<\u00a0.05). Finally, rs5275, rs4648298, and rs61764370 did not show significant associations. In conclusion, minor allele of rs3025039, rs3212986, and rs712 polymorphisms increases the risk of CRC in the East Asian population, and heterozygote model of rs731236 polymorphism shows protective effect in the Middle East population. In Europeans, the minor allele of rs17281995 may increase the risk of CRC, while rs712 may have a protective effect. Further analysis based on population stratifications should be considered in future 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BUON","nlmid":"100883428"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":124418903,"pmid_entry_id":30831803,"pmid_author_id":52904259,"position":7},{"pmid_author_map_id":124418902,"pmid_entry_id":30831803,"pmid_author_id":51681730,"position":6},{"pmid_author_map_id":124418901,"pmid_entry_id":30831803,"pmid_author_id":52904258,"position":5},{"pmid_author_map_id":124418900,"pmid_entry_id":30831803,"pmid_author_id":51681729,"position":4},{"pmid_author_map_id":124418899,"pmid_entry_id":30831803,"pmid_author_id":50846457,"position":3},{"pmid_author_map_id":124418898,"pmid_entry_id":30831803,"pmid_author_id":52904257,"position":2},{"pmid_author_map_id":124418897,"pmid_entry_id":30831803,"pmid_author_id":52904256,"position":1}],"pmid_abstract":[{"pmid_abstract_id":34993375,"pmid_entry_id":30831803,"abstract_type":"CONCLUSION","abstract_text":"Polymorphism rs712 in KRAS gene was protective factor associated with susceptibility for BC in this sample from Mexican population."},{"pmid_abstract_id":34993372,"pmid_entry_id":30831803,"abstract_type":"PURPOSE","abstract_text":"The rs712 polymorphism in a let-7 microRNA-binding KRAS gene has been associated with different types of cancer, however these associations have been inconsistent. The purpose of this study was to determine the association between rs712 polymorphism in a let-7 microRNA-binding KRAS gene comparing breast cancer (BC) patients with healthy subjects from Mexican population."},{"pmid_abstract_id":34993373,"pmid_entry_id":30831803,"abstract_type":"METHODS","abstract_text":"The genotyping of the rs712 polymorphism was performed by polymerase chain reaction (PCR) in 437 BC patients and 414 healthy women."},{"pmid_abstract_id":34993374,"pmid_entry_id":30831803,"abstract_type":"RESULTS","abstract_text":"The observed frequencies of the rs712 polymorphism indicated an associated protective factor for BC in the dominant GT+TT model [odds ratio (OR) 0.70, 95% confidence interval (CI) 0.51-0.97, p=0.040). An association between genotype and BC patients was evident in chemotherapy response (allele GT, OR 0.032, 95% CI 0.002-0.505, p=0.014), partial chemotherapy response (genotype GT, OR 0.023, 95% CI 0.001-0.419, p=0.011), and gastric and hematological toxicity (genotype GT, OR 0.115, 95% CI 0.028-0.473, p=0.003), Luminal A BC patients with gastric and hematological toxicity (genotype TT, OR 0.236, 95% CI 0.069-0.805, p=0.021) and tobacco consumption (genotype TT, OR 0.283, 95% CI 0.001-0.802, p=0.037) and Luminal B with metastatic lymph node (genotype GT, OR 0.241, 95% CI 0.093-0.626, p=0.003)."}]},{"pmid_author":[{"pmid_author_id":50846457,"last_name":"Eduardo Figuera","first_name":"Luis","initials":"L","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"7685e17dfdd2bf4f76fc3c8dc493e112"},{"pmid_author_id":51681729,"last_name":"Z\u00fa\u00f1iga Gonz\u00e1lez","first_name":"Guillermo Mois\u00e9s","initials":"GM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"8521ed995d2a1cd0cc539734668d9b3d"},{"pmid_author_id":51681730,"last_name":"Puebla P\u00e9rez","first_name":"Ana Mar\u00eda","initials":"AM","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"b0f79479c206a04a799914a33f61c691"},{"pmid_author_id":52904256,"last_name":"Patricia Gallegos-Arreola","first_name":"Martha","initials":"M","is_valid_author":"Y","pmid_instit_id":22296465,"orcid_id":null,"md5_hash":"a7aa3d6d5c1fd1aa2030faef64eb1b10"},{"pmid_author_id":52904257,"last_name":"Brise\u00f1o Zuno","first_name":"Carlos Jovany","initials":"CJ","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"ffc67036cbb6800eed221f24e80d888e"},{"pmid_author_id":52904258,"last_name":"Perales Mederos","first_name":"Carlos Iv\u00e1n","initials":"CI","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c6880823b18883770218a4a5a6c556c6"},{"pmid_author_id":52904259,"last_name":"Rosales Reynoso","first_name":"M\u00f3nica Alejandra","initials":"MA","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"c31e97b5d717f94c05d33c5ebe002c03"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":22296465,"instit_name":"Genetics Division, Western Biomedical Research Center, Mexican Institute of Social Security, Guadalajara, Jalisco, M\u00e9xico.","instit_hash":"f25b080bca6efbb3624be11fcf58bc40"}]}]},{"RECORD":{"pmid_entry_id":31923902,"pmid":33390813,"publication_date":"2021-01-01 00:00:00","title":"Association analysis between the interaction of RAS family genes mutations and papillary thyroid carcinoma in the Han Chinese population.","doi":"10.7150\/ijms.50026","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":19975,"month_1910":-1332,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":19975,"journal_name":"International journal of medical sciences","journal_abbr":"Int J Med Sci","issn_print":"","issn_online":"1449-1907","iso_abbr":"Int J Med Sci","nlmid":"101213954"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":131156770,"pmid_entry_id":31923902,"pmid_author_id":31026480,"position":7},{"pmid_author_map_id":131156769,"pmid_entry_id":31923902,"pmid_author_id":57705565,"position":6},{"pmid_author_map_id":131156768,"pmid_entry_id":31923902,"pmid_author_id":48359390,"position":5},{"pmid_author_map_id":131156767,"pmid_entry_id":31923902,"pmid_author_id":48359388,"position":4},{"pmid_author_map_id":131156766,"pmid_entry_id":31923902,"pmid_author_id":46813469,"position":3},{"pmid_author_map_id":131156765,"pmid_entry_id":31923902,"pmid_author_id":57705564,"position":2},{"pmid_author_map_id":131156764,"pmid_entry_id":31923902,"pmid_author_id":56851794,"position":1}],"pmid_abstract":[{"pmid_abstract_id":36959070,"pmid_entry_id":31923902,"abstract_type":"Text","abstract_text":"Papillary thyroid carcinoma (PTC) is the major subtype of thyroid cancer, accounting for 75%-85% of all thyroid malignancies. This study aimed to identify the association between the interactions of single nucleotide polymorphisms (SNPs) in RAS family genes and PTC in the Han Chinese population, to provide clues to the pathogenesis and potential therapeutic targets for PTC. Hap Map and NCBI-db SNP databases were used to retrieve SNPs. Haploview 4.2 software was used to filter SNPs based on specific parameters, six SNPs of RAS gene (KRAS-rs12427141, KRAS-rs712, KRAS-rs7315339, HRAS-rs12628, NRAS-rs14804 and NRAS-rs2273267) were genotyped by matrix-assisted laser desorption\/ionization time of flight mass spectrometry (MALDI-TOF-MS) in 673 PTC patients and 657 healthy controls, the interactive effect was evaluated by crossover analysis, logistic regression and GMDR software. We found that genetic mutation in rs712 have significant associations with PTC risk after Bonferroni correction (<0.001). The interaction between KRAS-rs12427141 and HRAS-rs12628 increased the risk of PTC (U=-2.119, <0.05), the interaction between KRAS-rs2273267 and HRAS-rs7315339 reduced the risk of PTC (U=2.195, <0.05). GMDR analysis showed that the two-factor model (KRAS-rs712, NRAS-rs2273267) was the best (=0.0107). Summarily, there are PTC-related interactions between RAS family genes polymorphisms in the Han Chinese population."}]},{"pmid_author":[{"pmid_author_id":31026480,"last_name":"Yu","first_name":"Qiong","initials":"Q","is_valid_author":"Y","pmid_instit_id":12928231,"orcid_id":null,"md5_hash":"1d282d77a4a8cb64615bf295b695b3be"},{"pmid_author_id":46813469,"last_name":"Sun","first_name":"Yaoyao","initials":"Y","is_valid_author":"Y","pmid_instit_id":12928231,"orcid_id":null,"md5_hash":"3ec29e3b5092acad1dd79c9089614bf6"},{"pmid_author_id":48359388,"last_name":"Zhang","first_name":"Mingyuan","initials":"M","is_valid_author":"Y","pmid_instit_id":12928231,"orcid_id":null,"md5_hash":"6c44c4dec6c9e396ee01fa72ced683fb"},{"pmid_author_id":48359390,"last_name":"Chen","first_name":"Xin","initials":"X","is_valid_author":"Y","pmid_instit_id":12928231,"orcid_id":null,"md5_hash":"9c5925900cce254dded849503f1d6a19"},{"pmid_author_id":56851794,"last_name":"Jin","first_name":"Mengdi","initials":"M","is_valid_author":"Y","pmid_instit_id":1,"orcid_id":null,"md5_hash":"d14047b10d9cb4ccd57e16b48dc14726"},{"pmid_author_id":57705564,"last_name":"Li","first_name":"Zhijun","initials":"Z","is_valid_author":"Y","pmid_instit_id":12928231,"orcid_id":null,"md5_hash":"0285cf69972ee6173c2f251f32ac2069"},{"pmid_author_id":57705565,"last_name":"Zhao","first_name":"Hongguang","initials":"H","is_valid_author":"Y","pmid_instit_id":24185178,"orcid_id":null,"md5_hash":"d42cae3f903072e0d2e6b133e45c13d3"}]},{"pmid_instit":[{"pmid_instit_id":1,"instit_name":"NULL","instit_hash":"6c3e226b4d4795d518ab341b0824ec29"},{"pmid_instit_id":12928231,"instit_name":"Department of Epidemiology and Biostatistics, School of Public Health, Jilin University, Changchun 130021, China.","instit_hash":"fc0c91d41711aa790f530a06f329ee71"},{"pmid_instit_id":24185178,"instit_name":"Nuclear Medicine Department, First Hospital of Jilin University, Changchun 130021, China.","instit_hash":"d1299174e2c89850f6128413b2b91abe"}]}]},{"RECORD":{"pmid_entry_id":33336990,"pmid":34828284,"publication_date":"2021-10-23 00:00:00","title":"Associations of Polymorphisms Localized in the 3'UTR Regions of the KRAS<\/i>, NRAS<\/i>, MAPK1<\/i> Genes with Laryngeal Squamous Cell Carcinoma.","doi":"10.3390\/genes12111679","volume":null,"issue":null,"pages":null,"pmid_status":"epublish","pmid_journal_id":26542,"month_1910":-1341,"full_text_date":null,"TABLE":"pmid_entry"},"PARENT":[{"pmid_journal":[{"pmid_journal_id":26542,"journal_name":"Genes","journal_abbr":"Genes (Basel)","issn_print":"","issn_online":"2073-4425","iso_abbr":"Genes (Basel)","nlmid":"101551097"}]}],"CHILD":[{"pmid_author_map":[{"pmid_author_map_id":140026861,"pmid_entry_id":33336990,"pmid_author_id":63922027,"position":7},{"pmid_author_map_id":140026860,"pmid_entry_id":33336990,"pmid_author_id":63922026,"position":6},{"pmid_author_map_id":140026859,"pmid_entry_id":33336990,"pmid_author_id":63922025,"position":5},{"pmid_author_map_id":140026858,"pmid_entry_id":33336990,"pmid_author_id":63922024,"position":4},{"pmid_author_map_id":140026857,"pmid_entry_id":33336990,"pmid_author_id":63922023,"position":3},{"pmid_author_map_id":140026856,"pmid_entry_id":33336990,"pmid_author_id":63922022,"position":2},{"pmid_author_map_id":140026855,"pmid_entry_id":33336990,"pmid_author_id":63922021,"position":1}],"pmid_abstract":[{"pmid_abstract_id":39494659,"pmid_entry_id":33336990,"abstract_type":"BACKGROUND","abstract_text":"Genetic variations, localized in the 3' untranslated region (UTR) in mitogen-activated protein kinase (MAPK) pathway-related genes, may alter the transcription and impact the pathogenesis of laryngeal squamous cell carcinoma (LSCC). The present study investigated the associations of single-nucleotide polymorphisms (SNP), localized in the 3'UTR) of the , and genes with LSCC risk and clinicopathological features."},{"pmid_abstract_id":39494660,"pmid_entry_id":33336990,"abstract_type":"METHODS","abstract_text":"Genomic DNA and clinical data were collected from 327 adult men with LSCC. The control group was formed from 333 healthy men. Genotyping of the SNPs was performed using TaqMan SNP genotyping assays. Five , and polymorphisms were analyzed. All studied genotypes were in Hardy-Weinberg equilibrium and had the same allele distribution as the 1000 Genomes project Phase 3 dataset for the European population."},{"pmid_abstract_id":39494661,"pmid_entry_id":33336990,"abstract_type":"RESULTS","abstract_text":"Significant associations of the studied SNPs with reduced LSCC risk were observed between rs14804 major genotype CC. Significant associations of the studied SNPs with clinicopathologic variables were also observed between rs14804 minor T allele and advanced tumor stage and positive lymph node status. SNP of rs9340 was associated with distant metastasis. Moreover, haplotype analysis of two SNPs rs712 and rs7973450 revealed that TG haplotype was associated with positive lymph node status in LSCC patients."},{"pmid_abstract_id":39494662,"pmid_entry_id":33336990,"abstract_type":"CONCLUSIONS","abstract_text":"According to the present study, 3'UTR SNP in the and genes may contribute to the identifications of patients at higher risk of LSCC lymph node and distant metastasis development."}]},{"pmid_author":[{"pmid_author_id":63922021,"last_name":"Insodaite","first_name":"Ruta","initials":"R","is_valid_author":"Y","pmid_instit_id":26560241,"orcid_id":null,"md5_hash":"ae18506612bac2730e4d6f357f8183ce"},{"pmid_author_id":63922022,"last_name":"Smalinskiene","first_name":"Alina","initials":"A","is_valid_author":"Y","pmid_instit_id":26560241,"orcid_id":null,"md5_hash":"11b3e4f1eb435a377d279e28a6936b12"},{"pmid_author_id":63922023,"last_name":"Liutkevicius","first_name":"Vykintas","initials":"V","is_valid_author":"Y","pmid_instit_id":26560242,"orcid_id":null,"md5_hash":"af49c26e5632e68459d3b062902dde78"},{"pmid_author_id":63922024,"last_name":"Ulozas","first_name":"Virgilijus","initials":"V","is_valid_author":"Y","pmid_instit_id":26560242,"orcid_id":null,"md5_hash":"6dc4e8a6b0cb103f727dfec885b8bc61"},{"pmid_author_id":63922025,"last_name":"Poceviciute","first_name":"Roberta","initials":"R","is_valid_author":"Y","pmid_instit_id":26560241,"orcid_id":null,"md5_hash":"4f97ba4a23b9b1be2a6e1acec88919e0"},{"pmid_author_id":63922026,"last_name":"Bielevicius","first_name":"Arunas","initials":"A","is_valid_author":"Y","pmid_instit_id":26560241,"orcid_id":null,"md5_hash":"f4f52bbea7d72bd660b924749eb355ce"},{"pmid_author_id":63922027,"last_name":"Kucinskas","first_name":"Laimutis","initials":"L","is_valid_author":"Y","pmid_instit_id":26560241,"orcid_id":null,"md5_hash":"15ea5ceb4aa9b4d2310e7a052516eda3"}]},{"pmid_instit":[{"pmid_instit_id":26560241,"instit_name":"Institute 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American College of Medical Genetics and Genomics (ACMG) previously developed guidance for the interpretation of sequence variants.(1) In the past decade, sequencing technology has evolved rapidly with the advent of high-throughput next-generation sequencing. By adopting and leveraging next-generation sequencing, clinical laboratories are now performing an ever-increasing catalogue of genetic testing spanning genotyping, single genes, gene panels, exomes, genomes, transcriptomes, and epigenetic assays for genetic disorders. By virtue of increased complexity, this shift in genetic testing has been accompanied by new challenges in sequence interpretation. In this context the ACMG convened a workgroup in 2013 comprising representatives from the ACMG, the Association for Molecular Pathology (AMP), and the College of American Pathologists to revisit and revise the standards and guidelines for the interpretation of sequence variants. The group consisted of clinical laboratory directors and clinicians. This report represents expert opinion of the workgroup with input from ACMG, AMP, and College of American Pathologists stakeholders. These recommendations primarily apply to the breadth of genetic tests used in clinical laboratories, including genotyping, single genes, panels, exomes, and genomes. This report recommends the use of specific standard terminology-\"pathogenic,\" \"likely pathogenic,\" \"uncertain significance,\" \"likely benign,\" and \"benign\"-to describe variants identified in genes that cause Mendelian disorders. Moreover, this recommendation describes a process for classifying variants into these five categories based on criteria using typical types of variant evidence (e.g., population data, computational data, functional data, segregation data). Because of the increased complexity of analysis and interpretation of clinical genetic testing described in this report, the ACMG strongly recommends that clinical molecular genetic testing should be performed in a Clinical Laboratory Improvement Amendments-approved laboratory, with results interpreted by a board-certified clinical molecular geneticist or molecular genetic pathologist or the equivalent."}]},{"pmid_author":[{"pmid_author_id":26289546,"last_name":"Bale","first_name":"Sherri","initials":"S","is_valid_author":"Y","pmid_instit_id":9480569,"orcid_id":null,"md5_hash":"9580e2f7e3908f5e939b070a5ddeb0c2"},{"pmid_author_id":27591935,"last_name":"Richards","first_name":"Sue","initials":"S","is_valid_author":"Y","pmid_instit_id":8699018,"orcid_id":null,"md5_hash":"b69c990072417ff742af77b84d932551"},{"pmid_author_id":27591936,"last_name":"Aziz","first_name":"Nazneen","initials":"N","is_valid_author":"Y","pmid_instit_id":11314361,"orcid_id":null,"md5_hash":"b1947fc3bbeed694b7ff9a57bd9442fc"},{"pmid_author_id":27591937,"last_name":"Bick","first_name":"David","initials":"D","is_valid_author":"Y","pmid_instit_id":11314362,"orcid_id":null,"md5_hash":"13425730d08598b6a3f6380ce2bf71b8"},{"pmid_author_id":27591938,"last_name":"Das","first_name":"Soma","initials":"S","is_valid_author":"Y","pmid_instit_id":11314363,"orcid_id":null,"md5_hash":"0e4d2dc2dd59e8b25d57d121d7ba9e9c"},{"pmid_author_id":27591939,"last_name":"Gastier-Foster","first_name":"Julie","initials":"J","is_valid_author":"Y","pmid_instit_id":11314364,"orcid_id":null,"md5_hash":"059f2c374893bfc4f09813e39b7bb4da"},{"pmid_author_id":27591940,"last_name":"Grody","first_name":"Wayne 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USA.","instit_hash":"b5ab499ef1f75f51ce179baaf2096fcc"},{"pmid_instit_id":11314366,"instit_name":"Department of Human Genetics, Emory Genetics Laboratory, Emory University, Atlanta, Georgia, USA.","instit_hash":"94eaa12a8b3bcd98c5b32a721027e38e"},{"pmid_instit_id":11314367,"instit_name":"Department of Pathology, ARUP Institute for Clinical and Experimental Pathology, University of Utah, Salt Lake City, Utah, USA.","instit_hash":"4ac29d381e5ccfbd757f03e9e35699e2"},{"pmid_instit_id":11314368,"instit_name":"Department of Pediatrics, Molecular Genetics Laboratory, Children's Hospital Colorado, University of Colorado Anschutz Medical School, Denver, Colorado, USA.","instit_hash":"118df5bd869b58c1421fcc3e42a3735e"},{"pmid_instit_id":11314369,"instit_name":"Partners Laboratory for Molecular Medicine and Department of Pathology, Brigham & Women's Hospital and Harvard Medical School, Boston, Massachusetts, 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2015 American College of Medical Genetics and Genomics-Association for Molecular Pathology (ACMG-AMP) guidelines were a major step toward establishing a common framework for variant classification. In practice, however, several aspects of the guidelines lack specificity, are subject to varied interpretations, or fail to capture relevant aspects of clinical molecular genetics. A simple implementation of the guidelines in their current form is insufficient for consistent and comprehensive variant classification.MethodsWe undertook an iterative process of refining the ACMG-AMP guidelines. We used the guidelines to classify more than 40,000 clinically observed variants, assessed the outcome, and refined the classification criteria to capture exceptions and edge cases. During this process, the criteria evolved through eight major and minor revisions.ResultsOur implementation: (i) separated ambiguous ACMG-AMP criteria into a set of discrete but related rules with refined weights; (ii) grouped certain criteria to protect against the overcounting of conceptually related evidence; and (iii) replaced the \"clinical criteria\" style of the guidelines with additive, semiquantitative criteria.ConclusionSherloc builds on the strong framework of 33 rules established by the ACMG-AMP guidelines and introduces 108 detailed refinements, which support a more consistent and transparent approach to variant classification."}]},{"pmid_author":[{"pmid_author_id":37666482,"last_name":"Nykamp","first_name":"Keith","initials":"K","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"04272f3193cecd4c22f301e418758359"},{"pmid_author_id":37666483,"last_name":"Anderson","first_name":"Michael","initials":"M","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"562335868814f8830c2851806db93c31"},{"pmid_author_id":37666484,"last_name":"Powers","first_name":"Martin","initials":"M","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"496c967cdd163aaf8c8b45031d06a3d4"},{"pmid_author_id":37666485,"last_name":"Garcia","first_name":"John","initials":"J","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"db70a46c6592cc833eb3ddd2fb4a4c38"},{"pmid_author_id":37666486,"last_name":"Herrera","first_name":"Blanca","initials":"B","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"bdee13030c40e97e7941f6b60afebc07"},{"pmid_author_id":37666487,"last_name":"Ho","first_name":"Yuan-Yuan","initials":"YY","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"4800f403db6bcd645613c96609baf1ad"},{"pmid_author_id":37666488,"last_name":"Kobayashi","first_name":"Yuya","initials":"Y","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"286ce435d252e7ea11235ef9d7d871c5"},{"pmid_author_id":37666489,"last_name":"Patil","first_name":"Nila","initials":"N","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"bfb8c7d79acf40e86d3dfa51594ee7c3"},{"pmid_author_id":37666490,"last_name":"Thusberg","first_name":"Janita","initials":"J","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"ec7b9eed580b4f306979229893991165"},{"pmid_author_id":37666491,"last_name":"Westbrook","first_name":"Marjorie","initials":"M","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"acf49516c10e50fcee616946f8606ee7"},{"pmid_author_id":37666492,"last_name":"Topper","first_name":"Scott","initials":"S","is_valid_author":"Y","pmid_instit_id":13649910,"orcid_id":null,"md5_hash":"e883ae800230ee0aa54bdd5ab3ae3973"}]},{"pmid_instit":[{"pmid_instit_id":13649910,"instit_name":"Invitae 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echo "INSERTING ".$TABLE." - ".count($TABLE_DATA)." records \n"; + echo "PROCESSING ".$TABLE." - ".count($TABLE_DATA)." records \n"; insert_table_data($schema,$TABLE,$TABLE_DATA,$ID_MAP); - echo "END INSERTING ".$TABLE." - ".count($TABLE_DATA)." records \n"; + echo "END PROCESSING ".$TABLE." - ".count($TABLE_DATA)." records \n"; $TABLE_DATA=array(); } /// We insert the remaining data - echo "INSERTING ".$TABLE." - ".count($TABLE_DATA)." records \n"; + echo "PROCESSING ".$TABLE." - ".count($TABLE_DATA)." records \n"; insert_table_data($schema,$TABLE,$TABLE_DATA,$ID_MAP); - echo "END INSERTING ".$TABLE." - ".count($TABLE_DATA)." records \n"; + echo "END PROCESSING ".$TABLE." - ".count($TABLE_DATA)." records \n"; } fclose($fp); @@ -117,7 +117,7 @@ function process_document($schema,$FILE_NAME) //print_R($ID_MAP); /// Rollback the transaction if issue $DB_CONN->rollBack(); - die($e->getMessage()); + echo ($e->getMessage()); } } @@ -136,6 +136,7 @@ function quoteValues(&$VALUES) function insert_table_data($schema,$TABLE_NAME,&$TABLE_DATA,&$ID_MAP) { + global $DEBUG; global $NOT_NULL; global $JSON_PARENT_FOREIGN; global $PRIMARY_KEYS; @@ -156,16 +157,10 @@ function insert_table_data($schema,$TABLE_NAME,&$TABLE_DATA,&$ID_MAP) /// We need to know the foreign keys for that table $PARENT_RULES=&$JSON_PARENT_FOREIGN['PARENT'][$schema.'.'.$TABLE_NAME]; - - $MAX_PK=-1; - $PK=null; - - /// Loop through the data - foreach ($TABLE_DATA as &$ENTRY) - { - - /// First things first, we need to convert any foreign key values defined in the file to the primary key of the parent table - if ($PARENT_RULES!=null) + /// First things first, we need to convert any foreign key values defined in the file to the primary key of the parent table + if ($PARENT_RULES!=null) + /// Loop through the data + foreach ($TABLE_DATA as &$ENTRY) { /// Therefore we need to check the PARENT_RULES array foreach ($PARENT_RULES as $TBL_COL=>&$PARENT_INFO) @@ -185,163 +180,284 @@ function insert_table_data($schema,$TABLE_NAME,&$TABLE_DATA,&$ID_MAP) } } - //print_R($ENTRY); + /// Now we break it down into chunks of 1000 records to get inserted + $CHUNKS=array_chunk($TABLE_DATA,1000); + + foreach ($CHUNKS as &$CHUNK) insert_table_data_chunk($CHUNK,$schema,$TABLE_NAME,$ID_MAP); +} - /// Now we check if the record exists: - $query="SELECT * FROM ".$schema.'.'.$TABLE_NAME." WHERE "; - /// To do that we use the unique columns for that table: - foreach ($KEYS[$TABLE_NAME] as $KEY) + + +function insert_table_data_chunk(&$TABLE_DATA,$schema,$TABLE_NAME,&$ID_MAP) +{ + global $NOT_NULL; + global $JSON_PARENT_FOREIGN; + global $PRIMARY_KEYS; + global $KEYS; + global $DEBUG; + + /// Getting the primary keys specific to that table + $PRIMARY_KEY=$PRIMARY_KEYS[$schema.'.'.$TABLE_NAME]; + + /// And the keys + $TABLE_KEYS=&$KEYS[$TABLE_NAME]; + + + /// The first step is to check if the record is already in the database or not + /// A challenge arise when some of the values are NULL since in a group query the NULL is not recognized + /// Therefore, we need to do multiple queries depending on which set of column is null or not + /// Example. Given 3 columns A B and C, in which B and C are nullable + /// We can have 4 cases: + /// A B C + /// 1 1 1 => (A,B,C) IN ((x,x,x),(x,x,x),(x,x,x)) + /// 1 1 0 => (A,B) IN ((x,x),(x,x),(x,x)) AND C IS NULL + /// 1 0 1 => (A,C) IN ((x,x),(x,x),(x,x)) AND B IS NULL + /// 1 0 0 => (A) IN ((x),(x),(x)) AND B IS NULL AND C IS NULL + /// That's what query_params is about. It's an array with the query head as key and the values as values + + + $QUERY_PARAMS=array(); + + /// ID_PARAMS is used to map the different values represented as a hash to the position in the TABLE_DATA array + /// This will help us to know which record we are dealing with when we get the result of the query + $ID_PARAMS=array(); + + foreach ($TABLE_DATA as $ENTRY_POS=>&$ENTRY) + { + /// The string ID is used to store the values of the record so we can hash it + $STR_ID=''; + + /// Temporary array to store the values + $VALUES=array(); + foreach ($TABLE_KEYS as $KEY) { if (!isset($ENTRY[$KEY]))die('Key '.$KEY.' not found in entry '.$TABLE_NAME); /// The value is NULL? Special case applies: + if ($ENTRY[$KEY]=='NULL') { - echo $schema.'.'.$TABLE_NAME.'.'.$KEY."\n"; - + // echo $schema.'.'.$TABLE_NAME.'.'.$KEY."\n"; + $STR_ID.='|||'; /// If the column is not nullable, then we need to replace it by either IS NULL or ='' depending on the type if (isset($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY])) { if ($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][0]) { - $query.=' '.$KEY."= '' AND "; + $VALUES[$KEY]="''"; } else { //echo "NOT\n"; - $query.=" (".$KEY." IS NULL "; - switch($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][1]) - { - case 'integer': - case 'bigint': - case 'smallint': - case 'numeric': - case 'real': - case 'double precision': - - break; - case 'timestamp': - case 'date': - - break; - default: - $query.=' OR '.$KEY." = ''"; - } - - - $query.=") AND "; + $sub_q="NULL"; + $VALUES[$KEY]=$sub_q; } } else { - $query.=' '.$KEY."= NULL AND "; + $VALUES[$KEY]="NULL"; } } /// Otherwise easy enough $KEY=>$VALUE - else $query.=" ".$KEY."='".str_replace("'","''",$ENTRY[$KEY])."' AND "; + else + { + $VALUES[$KEY]="'".str_replace("'","''",$ENTRY[$KEY])."'"; + $STR_ID.=$ENTRY[$KEY]."|||"; + } + } + + /// Now we need to separate the values in 2 arrays, one for the values that are not NULL and one for the values that are NULL + $STR_NULL=array(); + $STR_HEAD=array();$VALUE_HEAD=array(); + foreach ($VALUES as $KEY_V=>&$VALUE_V) + { + if ($VALUE_V!='NULL') + { + $STR_HEAD[]=$KEY_V; + $VALUE_HEAD[]=$VALUE_V; + } + else $STR_NULL[]=$KEY_V.' IS NULL'; } - /// Remove the last AND with space - $query=substr($query,0,-4); - /// We run the query + /// We build the query head + $STR_LEN_NULL=implode(' AND ',$STR_NULL); + if ($STR_LEN_NULL!='')$STR_LEN_NULL=$STR_LEN_NULL.' AND '; + $STR_KEY_HEAD="SELECT * FROM ".$schema.'.'.$TABLE_NAME." WHERE ".$STR_LEN_NULL.' ('.implode(",",$STR_HEAD).') IN ('; + + /// We store the query head and the values + $QUERY_PARAMS[$STR_KEY_HEAD][]='('.implode(",",$VALUE_HEAD).")"; + + /// We store the position of the record in the TABLE_DATA array + $ID_PARAMS[md5($STR_ID)]=$ENTRY_POS; + } + + if ($DEBUG)print_R($QUERY_PARAMS); + + /// Now we run the queries + /// N_FOUND is used to count the number of records found + $N_FOUND=0; + foreach ($QUERY_PARAMS as $STR_KEY_HEAD=>&$QUERY_VALUES) + { + /// Merge the query head with the values + $query=$STR_KEY_HEAD.' '.implode(",",$QUERY_VALUES).")"; $res=runQuery($query); - if ($res===false)throw new Exception("Unable to run query ".$query); - - /// No results! We need to insert the - if ($res==array()) + + + /// The record exists, we store the primary key + foreach ($res as &$line) { - /// So we need to know the primary key for that table - /// Get it's max value so we can increment it - if ($MAX_PK==-1) + + /// So first we need to create the hash based on the value + $str_k=''; + foreach ($TABLE_KEYS as $KEY) { - if (count($PRIMARY_KEY)==1) - { - echo "SELECT MAX(".$PRIMARY_KEY[0].") m FROM ".$schema.'.'.$TABLE_NAME."\n"; - $res=runQuery("SELECT MAX(".$PRIMARY_KEY[0].") m FROM ".$schema.'.'.$TABLE_NAME); - if ($res[0]['m']!='') $MAX_PK=$res[0]['m']; - else $MAX_PK=0; - $PK=$PRIMARY_KEY[0]; - } - echo "MAX PK : ".$MAX_PK."\t".$PK; + if ($line[$KEY]=='NULL')$str_k.='|||'; + else + $str_k.=$line[$KEY]."|||"; } - - /// Now we build the query - $query="INSERT INTO ".$schema.'.'.$TABLE_NAME." ("; - /// By looping through the columns - foreach ($ENTRY as $KEY=>$VALUE) + $hash=md5($str_k); + //echo implode("\t",$line)."\t".$hash."\n"; + /// So we can find the corresponding position of the record in TABLE_DATA + if (!isset($ID_PARAMS[$hash]))throw new Exception("Unable to find entry in ID_PARAMS"); + + /// Now technically, we should not have multiple records found, so if DB_ID is already set, we throw an exception + if (isset($TABLE_DATA[$ID_PARAMS[$hash]]['DB_ID'])) + {print_R($TABLE_DATA[$ID_PARAMS[$hash]]); + throw new Exception("Multiple records found"); + } + + /// Reference to the record + $ENTRY=&$TABLE_DATA[$ID_PARAMS[$hash]]; + // echo "\t=>".$ID_PARAMS[$hash]."\t==>".implode("\t",$ENTRY)."\n"; + /// We store the primary key + $ENTRY['DB_ID']=$line[$PRIMARY_KEY[0]]; + + /// Then we need to store the primary key in the ID_MAP, + /// which will help mapping for a given table the ID in the file to the ID in the database + $str_k=''; + $db_k=''; + foreach ($PRIMARY_KEY as $K) { - $query.=$KEY.","; + $str_k.=$ENTRY[$K].'||'; + $db_k.=$line[$K].'||'; } - $query=substr($query,0,-1).") VALUES ("; + if ($DEBUG)echo $TABLE_NAME.' '.substr($str_k,0,-2).' '.substr($db_k,0,-2)."\n"; + $ID_MAP[$TABLE_NAME][substr($str_k,0,-2)]=substr($db_k,0,-2); + + ++$N_FOUND; + } + } + + echo "FOUND ".$N_FOUND."/".count($TABLE_DATA)." records\n"; + + /// All records are found, we can return as the job is done + if ($N_FOUND==count($TABLE_DATA))return; + + + //// So we need to know the primary key for that table + /// Get it's max value so we can increment it + $MAX_PK=-1; + $PK=null; + if (count($PRIMARY_KEY)==1) + { + if ($DEBUG) echo "SELECT MAX(".$PRIMARY_KEY[0].") m FROM ".$schema.'.'.$TABLE_NAME."\n"; + $res=runQuery("SELECT MAX(".$PRIMARY_KEY[0].") m FROM ".$schema.'.'.$TABLE_NAME); + if ($res[0]['m']!='') $MAX_PK=$res[0]['m']; + else $MAX_PK=0; + $PK=$PRIMARY_KEY[0]; + }else throw new Exception("Multiple primary keys for table ".$schema.'.'.$TABLE_NAME); + + if ($DEBUG)echo "MAX PK : ".$MAX_PK."\t".$PK; + + /// Now we build the single query with multiple insert records + $query="INSERT INTO ".$schema.'.'.$TABLE_NAME." ("; + /// The easiest is to create arrays of keys and values and then implode them + + $QUERY_KEYS=array(); + $RECORDS=array(); + + foreach ($TABLE_DATA as $ENTRY_POS=>&$ENTRY) + { + if (isset($ENTRY['DB_ID']))continue; + if ($DEBUG)print_R($ENTRY); + + /// First record to process, we extract the keys + if ($QUERY_KEYS==array()) + { + $QUERY_KEYS=array_keys($ENTRY); + } + + /// Then the values: + $VALUES=array(); + + foreach ($ENTRY as $KEY=>&$VALUE) + { + /// Special case for NULL values, we need to check if the column is nullable + /// and if it is, we need to replace it by NULL or '' - /// Then the values: - foreach ($ENTRY as $KEY=>$VALUE) + if ($ENTRY[$KEY]=='NULL') { - /// Special case for NULL values, we need to check if the column is nullable - /// and if it is, we need to replace it by NULL or '' - - if ($ENTRY[$KEY]=='NULL') + if (isset($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY])) { - if (isset($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY])) + //print_R($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY]); + if ($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][0]) { - //print_R($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY]); - if ($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][0]) - { - - $query.=" '' , "; - - } - else - { - if ($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][1])$query.="NULL,"; - else $query.="NULL,"; - } + $VALUES[]="''"; } else { - $query.='NULL,'; + if ($NOT_NULL[$schema.'.'.$TABLE_NAME.'.'.$KEY][1])$VALUES[]="NULL"; + else $VALUES[]="NULL"; } - } - else if ($PK!=null && $PK==$KEY) - { - $MAX_PK++; - $query.=$MAX_PK.","; - }else $query.="'".str_replace("'","''",$VALUE)."',"; + else $VALUES[]='NULL'; } - - /// Remove the last comma - $query=substr($query,0,-1).") RETURNING *"; - - echo $query."\n"; - - $res=runQuery($query); - - if ($res===false)throw new Exception("Unable to run query ".$query); - - /// We store the primary key of the record we just inserted - $ID_MAP[$TABLE_NAME][$ENTRY[$PK]]=$MAX_PK; - - } - else - { - - if (count($res)!=1) throw new Exception("Multiple entries found for ".$query); - /// The record exists, we store the primary key - $str_k=''; - $db_k=''; - foreach ($PRIMARY_KEY as $K) + else if ($PK!=null && $PK==$KEY) { - $str_k.=$ENTRY[$K].'||'; - $db_k.=$res[0][$K].'||'; - } - - $ID_MAP[$TABLE_NAME][substr($str_k,0,-2)]=substr($db_k,0,-2); + $MAX_PK++; + $VALUES[]=$MAX_PK; + }else $VALUES[]="'".str_replace("'","''",$VALUE)."'"; } + /// concatenate the values into a string and add that to the record array + $RECORDS[]="(".implode(",",$VALUES).")\n"; + } + if ($RECORDS==array())return; + /// Now we create the complete query and request to return everything + $query.=implode(",",$QUERY_KEYS).") VALUES ".implode(",",$RECORDS)." RETURNING *"; + + if ($DEBUG)echo $query."\n"; + + $res=runQuery($query); + if ($DEBUG)print_R($res); + if ($res===false)throw new Exception("Unable to run query ".$query); + + //print_r($ID_PARAMS); + foreach ($res as &$line) + { + if ($DEBUG)print_R($line); + /// Similar to the previous case, we need to create a hash to find the corresponding record in TABLE_DATA + $str_k=''; + foreach ($TABLE_KEYS as $KEY) + { + $str_k.=$line[$KEY]."|||"; + } + $hash=md5($str_k); + if (!isset($ID_PARAMS[$hash]))throw new Exception("Unable to find entry in ID_PARAMS"); + /// Reference to the record + $ENTRY=&$TABLE_DATA[$ID_PARAMS[$hash]]; + /// Then we need to store the primary key in the ID_MAP, + /// which will help mapping for a given table the ID in the file to the ID in the database + $ID_MAP[$TABLE_NAME][$ENTRY[$PK]]=$line[$PK]; + if ($DEBUG) + { + print_R($ENTRY); + echo $ENTRY[$PK].'>>>>'.$line[$PK]."\n"; + echo "#####\n"; + } } - + } @@ -349,6 +465,8 @@ function insert_table_data($schema,$TABLE_NAME,&$TABLE_DATA,&$ID_MAP) + + function search_record($schema,$table,$column,$list) { if ($list==array())return array();